Induction of multiple autoantibodies to islet cell antigens during treatment with interferon alpha for chronic hepatitis C [3]

Gut

Volumn 49, Issue 4, 2001, Pages 596-597

  Wasmuth, H E ^a   Stolte, C ^a   Geier, A ^a   Gartung, C ^a   Matern, S ^a  

^a RWTH AACHEN UNIVERSITY   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

ALPHA INTERFERON; ANTIVIRUS AGENT; AUTOANTIBODY; AUTOANTIGEN; CELL ANTIGEN; GLUCOSE; GLUTAMATE DECARBOXYLASE; INSULIN; INSULIN ANTIBODY; PANCREAS ISLET CELL ANTIBODY; PHOSPHATASE; STEROID 21 MONOOXYGENASE; VIRUS RNA;

ANTIBODY TITER; CHRONIC HEPATITIS; DISEASE PREDISPOSITION; FAMILY HISTORY; FOLLOW UP; GLUCOSE TOLERANCE TEST; HEPATITIS C; HUMAN; IMMUNOFLUORESCENCE; INSULIN DEPENDENT DIABETES MELLITUS; LETTER; PRIORITY JOURNAL;

ADULT; ANTIVIRAL AGENTS; AUTOANTIBODIES; DIABETES MELLITUS, TYPE 1; FEMALE; HEPATITIS C, CHRONIC; HUMANS; INTERFERON-ALPHA; ISLETS OF LANGERHANS; MALE; MIDDLE AGED; RISK FACTORS;

EID: 0034823156     PISSN: 00175749     EISSN: None     Source Type: Journal    
DOI: 10.1136/gut.49.4.596     Document Type: Letter

References (5)

1. Immunology in diabetes: An update
2. Only multiple autoantibodies to islet cells (ICA), insulin, GAD65, IA-2 and IA-2 predict immune-mediated (type 1) diabetes in relatives
3. Autoantibodies in diabetes mellitus: Diagnostic value
4. Autoimmunity against pancreatic islets and other tissues before and after interferon-alpha therapy in patients with hepatitis C virus chronic infection
5. Prediction of IDDM in the general population. Strategies based on combination of autoantibody markers