Effects of enterostatin (Val-Pro-Asp-Pro-Arg) on fat intake and blood levels of glucose and insulin in rats

Methods and Findings in Experimental and Clinical Pharmacology

Volumn 23, Issue 5, 2001, Pages 235-239

  Koizumi, M ^a   Sato, H ^a   Seguro, K ^a   Ide, H ^a   Morinaga, Y ^a   Kimura, S ^a  

^a Grad School of Human Life Science   (Japan)

Author keywords

Enterostatin; Fat intake; Satiety factor; Val Pro Asp Pro Arg (VPDPR)

Indexed keywords

ANOREXIGENIC AGENT; ENTEROSTATIN; GLUCOSE; INSULIN;

ANIMAL EXPERIMENT; ANIMAL MODEL; APPETITE; ARTICLE; BRAIN THIRD VENTRICLE; CONTROLLED STUDY; DOSE RESPONSE; FAT INTAKE; FEEDING BEHAVIOR; GLUCOSE BLOOD LEVEL; INSULIN BLOOD LEVEL; LIPID DIET; LOW FAT DIET; MALE; NONHUMAN; RAT; SATIETY;

EID: 0034783422     PISSN: 03790355     EISSN: None     Source Type: Journal    
DOI: 10.1358/mf.2001.23.5.662117     Document Type: Article

References (12)

1. Suppression of food intake by apolipoprotein A-IV is mediated through the central nervous system in rats
2. Pancreatic colipase
3. 86
4. Enterostatin suppresses food intake following injection into the third ventricle of rats
5. Enterostatin (Val-Pro-Asp-Pro-Arg), the activation peptide of procolipase, selectively reduces fat intake
6. Pancreatic procolipase propeptide, enterostatin, specifically inhibits fat intake
7. Differential inhibition of fat intake in two strains of rat by the peptide enterostatin
8. Adrenalectomy of the obese Zucker rat: Effects on the feeding response to enterostatin and specific mRNA levels
9. Effect of enterostatin in non-food-deprived rats with limited or continuous access to oil or sucrose
10. Chronic ingestion of dietary fat is a prerequisite for inhibition of feeding by enterostatin
11. + channels in taste receptor cells: Gustatory cues for dietary fat