Increased nuclear factor-κB and angiotensinogen gene expression in posttransplant recurrent focal segmental glomerulosclerosis

Transplantation

Volumn 70, Issue 7, 2000, Pages 1107-1110

  Schachter, Asher D ^a,b,c   Strehlau, Jurgen ^a,b   Zurakowski, David ^a,c   Vasconcellos, Lauro ^a,b   Kim, Yon Su ^a,b   Zheng, Xin Xiao ^a,b   Gunshin, Yoshimi ^a,c   Overstreet, Sandra L ^d   Benfield, Mark R ^d   Tejani, Amir ^e   Harmon, William E ^a,c   Herrin, John T ^a,c   Strom, Terry B ^a,c  

^a HARVARD MEDICAL SCHOOL   (United States)

^b BETH ISRAEL DEACONESS MEDICAL CENTER   (United States)

^c BOSTON CHILDREN S HOSPITAL   (United States)

^d University of Alabama at Birmingham   (United States)

^e Clinical Coordinating Center   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ANGIOTENSIN; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN;

ADULT; ARTICLE; CADAVER; CAUCASIAN; DISEASE ASSOCIATION; GENE EXPRESSION; GLOMERULOSCLEROSIS; GRAFT REJECTION; HUMAN; HUMAN TISSUE; KIDNEY BIOPSY; KIDNEY FAILURE; KIDNEY GRAFT; MAJOR CLINICAL STUDY; MALE; NEGRO; POSTOPERATIVE COMPLICATION; PRIORITY JOURNAL; RACE DIFFERENCE; RECIPIENT; RECURRENT DISEASE; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION;

EID: 0034667980     PISSN: 00411337     EISSN: None     Source Type: Journal    
DOI: 10.1097/00007890-200010150-00021     Document Type: Article

References (11)

1. Changing patterns in the histopathology of idiopathic nephrotic syndrome in children
2. Age and ethnicity affect the risk and outcome of focal segmental glomerulosclerosis
3. Recurrence of focal segmental glomerulosclerosis posttransplantation: A special report of the North American Pediatric Renal Transplant Cooperative Study
4. Nuclear factor-κB. A pivotal transcription factor in chronic inflammatory diseases
5. Angiotensinogen gene activation by angiotensin II is mediated by the rel A (nuclear factor-kappaB p65) transcription factor: One mechanism for the renin angiotensin system positive feedback loop in hepatocytes
6. Immunosuppression by glucocorticoids: Inhibition of NF-κB activity through induction of IκB synthesis
7. Quantitative detection of immune activation transcripts as a diagnostic tool in kidney transplantation
8. In vivo transfection of genes from renin and angiotensinogen into the glomerular cells induced phenotypic change of the mesangial cells and glomerular sclerosis
9. Recurrence of steroid-resistant nephrotic syndrome in kidney transplants is associated with increased acute renal failure and acute rejection
10. Protein overload stimulates RANTES production by proximal tubular cells depending on NF-kappa B activation