Muscular dystrophy: The worm turns to genetic disease

Current Biology

Volumn 10, Issue 21, 2000, Pages

  Chamberlain, Jeffrey S ^a   Benian, Guy M ^b  

^a UNIVERSITY OF MICHIGAN MEDICAL SCHOOL   (United States)

^b EMORY UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CAENORHABDITIS ELEGANS;

EID: 0034597588     PISSN: 09609822     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0960-9822(00)00768-5     Document Type: Review

References (18)

1. Isolation of candidate cDNA clones for portions of the Duchenne muscular dystrophy gene
2. Genetic suppression of dystrophin-related phenotypes in Caenorhabditis elegans
3. Muscle
4. Muscle: Structure, function, and development
5. A role for the dystrophin-glycoprotein complex as a transmembrane linker between laminin and actin
6. Contraction-induced injury to single permeabilized muscle fibers from mdx, transgenic mdx, and control mice
7. Role for α-dystrobrevin in the pathogenesis of dystrophin-dependent muscular dystrophies
8. γ-sarcoglycan deficiency leads to muscle membrane defects and apoptosis independent of dystrophin
9. Assembly of the dystrophin associated protein complex does not require the dystrophin C-terminal domain
10. Mutations in the Caenorhabditis elegans dystrophin-like gene dys-1 lead to hyperactivity and suggest a link with cholinergic transmission
11. Dystrobrevin- and dystrophin-like mutants display similar phenotypes in the nematode Caenorhabditis elegans
12. Flying through the Drosophila cytoskeletal genome
13. Dystrophins in vertebrates and invertebrates
14. The dynamics of dystroglycan
15. MyoD is required for myogenic stem cell function in adult skeletal muscle
16. Progress towards gene therapy of Duchenne muscular dystrophy
17. The artificial zinc finger coding gene 'Jazz' binds the utrophin promoter and activates transcription