A molecular element of neuronal response to lysolecithin-induced focal, experimental demyelination in rat

Neurobiology

Volumn 8, Issue 3-4, 2000, Pages 273-275

  Lovas, G ^a   Palkovits, M ^a   Komoly, S ^a  

^a NATIONAL INSTITUTE OF NEUROLOGICAL DISORDERS AND STROKE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

GENE PRODUCT; LYSOPHOSPHATIDYLCHOLINE; PROTEIN C JUN; PROTEIN FOS; TRANSCRIPTION FACTOR;

ANIMAL TISSUE; CONTROLLED STUDY; DEMYELINATION; EXPERIMENTAL MODEL; GENE EXPRESSION; HISTOPATHOLOGY; IMMUNOHISTOCHEMISTRY; MULTIPLE SCLEROSIS; NERVE CELL; NERVE INJURY; NONHUMAN; NOTE; ONCOGENE C JUN; PATHOGENESIS; PROTEIN EXPRESSION; RAT;

ANIMALS; BRAIN; DEMYELINATING DISEASES; GENES, JUN; LYSOPHOSPHATIDYLCHOLINES; NEURONS; RATS; RATS, SPRAGUE-DAWLEY;

EID: 0034480361     PISSN: 12168068     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Note

References (8)

1. Lysophosphatidyl choline-induced demyelination. A freeze-fracture study
2. Medial septal neurons express c-Jun but do not undergo DNA fragmentation after fimbria-fornix transections
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6. Axonal changes in chronic demyelinated cervical spinal cord plaques
7. Axonal transection in the lesions of multiple sclerosis
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