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Volumn 128, Issue 3, 2000, Pages 173-188
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Different cell death mechanisms and gene expression in human cells induced by pentachlorophenol and its major metabolite, tetrachlorohydroquinone
a b c a a |
Author keywords
Apoptosis; Cytotoxicity; Pentachlorophenol; Tetrachlorohydroquinone
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Indexed keywords
CELLULAR APOPTOSIS SUSCEPTIBILITY PROTEIN;
DNA FRAGMENT;
GENE PRODUCT;
HEAT SHOCK PROTEIN 70;
PENTACHLOROPHENOL;
PROTEIN BAX;
PROTEIN BCL 2;
TETRACHLOROHYDROQUINONE;
UNCLASSIFIED DRUG;
ANALYTIC METHOD;
APOPTOSIS;
ARTICLE;
BLADDER;
CELL DEATH;
CELL STRAIN;
CONCENTRATION RESPONSE;
CONTROLLED STUDY;
CYTOTOXICITY;
DECHLORINATION;
DNA STRAND BREAKAGE;
GENE EXPRESSION;
HUMAN;
HUMAN CELL;
LIVER CELL;
POLLUTION;
TISSUE SPECIFICITY;
APOPTOSIS;
BCL-2-ASSOCIATED X PROTEIN;
CARCINOGENS;
CELL DEATH;
CELL LINE;
CELLULAR APOPTOSIS SUSCEPTIBILITY PROTEIN;
DNA FRAGMENTATION;
ENVIRONMENTAL POLLUTANTS;
FLOW CYTOMETRY;
GENE EXPRESSION;
HELA CELLS;
HSP70 HEAT-SHOCK PROTEINS;
HUMANS;
HYDROQUINONES;
IN SITU NICK-END LABELING;
LIVER;
PENTACHLOROPHENOL;
PROTEINS;
PROTO-ONCOGENE PROTEINS;
PROTO-ONCOGENE PROTEINS C-BCL-2;
TUMOR CELLS, CULTURED;
URINARY BLADDER;
ANIMALIA;
RODENTIA;
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EID: 0034327693
PISSN: 00092797
EISSN: None
Source Type: Journal
DOI: 10.1016/S0009-2797(00)00194-0 Document Type: Article |
Times cited : (49)
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References (46)
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