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Volumn 57, Issue 6, 2000, Pages 789-790

Autotoxicity and Alzheimer disease

Author keywords

[No Author keywords available]

Indexed keywords

ACUTE PHASE PROTEIN; ANTIINFLAMMATORY AGENT; COMPLEMENT RECEPTOR; IMMUNOGLOBULIN RECEPTOR;

EID: 0034084677     PISSN: 00039942     EISSN: None     Source Type: Journal    
DOI: 10.1001/archneur.57.6.789     Document Type: Short Survey
Times cited : (39)

References (10)
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    • Neuronal expression of mRNAs for complement proteins of the classical pathway in Alzheimer brain
    • Shen Y, Li R, McGeer EG, McGeer PL. Neuronal expression of mRNAs for complement proteins of the classical pathway in Alzheimer brain. Brain Res. 1997;769:391-395.
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    • Smith MA, Sayre LM, Anderson VE, et al. Cytochemical demonstration of oxidative damage in Alzheimer-disease by immunochemical enhancement of the carbonyl reaction with 2,4-dinitrophenylhydrazine. J Histochem Cytochem. 1998:46:731-735.
    • (1998) J Histochem Cytochem. , vol.46 , pp. 731-735
    • Smith, M.A.1    Sayre, L.M.2    Anderson, V.E.3
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    • Risk of Alzheimer's disease and duration of NSAID use
    • Stewart WF, Kawas C, Corrada M, Metter EJ. Risk of Alzheimer's disease and duration of NSAID use. Neurology. 1997;48:626-632.
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    • Human heart generates complement proteins that are upregulated and activated after myocardial infarction
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.