Prolonged exposure of mouse macrophages to IFN-β suppresses transcription of the inducible nitric oxide synthase gene: Altered availability of transcription factor Stat1α

European Journal of Immunology

Volumn 30, Issue 6, 2000, Pages 1551-1561

  Gao, Jian Jun ^a,d   Filla, Michael B ^a   Lorsbach, Robert B ^a,e   Pace, Judith L ^a,c   Crespo, Ana ^a,b   Russell, Stephen W ^a   Murphy, William J ^a,c  

^a UNIVERSITY OF KANSAS SCHOOL OF MEDICINE   (United States)

^b UNIVERSITY OF PORTO   (Portugal)

^c UNIVERSITY OF KANSAS MEDICAL CENTER   (United States)

^d UNIVERSITY OF MISSOURI KANSAS CITY   (United States)

^e ST JUDE CHILDREN S RESEARCH HOSPITAL   (United States)

Author keywords

Cellular activation; Gene regulation; Inflammatory mediator; Lipopolysaccharide; Molecular biology

Indexed keywords

BETA INTERFERON; DIMER; INTERFERON REGULATORY FACTOR 1; NITRIC OXIDE SYNTHASE; NITRIC OXIDE SYNTHASE INHIBITOR; STAT1 PROTEIN; TRANSCRIPTION FACTOR;

ANIMAL CELL; ANTINEOPLASTIC ACTIVITY; ARTICLE; CELL ACTIVATION; CONTROLLED STUDY; GENE EXPRESSION; GENE REPRESSION; MACROPHAGE; MALE; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION; TRANSCRIPTION REGULATION;

ANIMALS; CELL NUCLEUS; CELLS, CULTURED; CULTURE MEDIA; CYTOTOXICITY, IMMUNOLOGIC; DNA-BINDING PROTEINS; ENHANCER ELEMENTS (GENETICS); INTERFERON REGULATORY FACTOR-1; INTERFERON-BETA; INTERFERON-GAMMA, RECOMBINANT; KINETICS; LIPOPOLYSACCHARIDES; MACROPHAGES; MALE; MICE; MICE, INBRED C3H; NF-KAPPA B; NITRIC OXIDE SYNTHASE; NITRIC OXIDE SYNTHASE TYPE II; PHOSPHOPROTEINS; PHOSPHORYLATION; PROMOTER REGIONS (GENETICS); STAT1 TRANSCRIPTION FACTOR; TIME FACTORS; TRANS-ACTIVATORS; TRANSCRIPTION, GENETIC; TUMOR CELLS, CULTURED;

EID: 0034036767     PISSN: 00142980     EISSN: None     Source Type: Journal    
DOI: 10.1002/1521-4141(200006)30:6<1551::AID-IMMU1551>3.0.CO;2-Y     Document Type: Article

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