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Evidence for the presence of autoantibodies to the collagen-like portion of Clq in Systemic Lupus Erythematosus
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Homocygous Clq deficiency causes glomerulonephritis associated with multiple apoptotic bodies
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Autoantigens targeted in systemic lupus erythematosus are clustered in two populations of surface structures on apoptotic keratinocytes
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Changes in antibodies to Clq predict renal relapses in Systemic Lupus Erythematosus
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Autoantibodies to the collagen-like region of Clq are strongly associated with classical pathway-mediated hypocomplementemia in systemic lupus erythematosus
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Association between ongoing anti-Clq antibody production in peripheral blood and proliferative nephritis in patients with active systemic lupus erythematosus
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Clq binds directly and specifically to surface blebs on apoptotic human keratinocytes: Complement deficiency and systemic lupus erythematosus revisited
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Predictive value of IgG autoantibodies against Clq for nephritis in systemic lupus erythematosus
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IgG auto-antibodies against Clq are correlated with nephritis, hypocomplementemia, and dsDNA antibodies in systemic lupus erythematosus
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Hypocomplementemic urticarial vasculitis syndrome (HUVS) or systemic lupus erythematosus (SLE)?
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Lack of occurrence of severe lupus nephritis in anti-Clq autoantibody negative patients
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The spectrum of apoptotic defects and clinical manifestations, including systemic lupus erythematosus, in humans with CD95 (Fas/APO-1) mutations
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