Recent advances in the genetics of childhood renal cancers: A report of the 3rd International Conference on the molecular and clinical genetics of childhood renal tumors, together with the Mitchell Ross Symposium on anaplastic and other high risk embryonal tumors of childhood 8-10th April 1999, Wistar Institute, Philadelphia, PA

Medical and Pediatric Oncology

Volumn 35, Issue 2, 2000, Pages 126-130

  Jones, Kathryn Pritchard ^a   Grundy, Paul E ^b   Coppes, Max J ^c  

^a INSTITUTE OF CANCER RESEARCH   (United Kingdom)

^b UNIVERSITY OF ALBERTA   (Canada)

^c UNIVERSITY OF CALGARY   (Canada)

Author keywords

[No Author keywords available]

Indexed keywords

ANAPLASTIC CARCINOMA; ANIRIDIA; CANCER CHEMOTHERAPY; CANCER GENETICS; CHILDHOOD CANCER; CONFERENCE PAPER; EMBRYONAL CARCINOMA; GENE MUTATION; GENETIC PREDISPOSITION; HETEROZYGOSITY LOSS; KIDNEY CANCER; MULTIDRUG RESISTANCE; NEPHROBLASTOMA; PEDIGREE; PRIORITY JOURNAL; PROGNOSIS; TREATMENT OUTCOME; TUMOR SUPPRESSOR GENE;

CARCINOMA, EMBRYONAL; CHILD; HEPARAN SULFATE PROTEOGLYCAN; HUMAN; KIDNEY NEOPLASMS; NEPHROBLASTOMA; RHABDOID TUMOR;

EID: 0033863114     PISSN: 00981532     EISSN: None     Source Type: Journal    
DOI: 10.1002/1096-911X(200008)35:2<126::AID-MPO8>3.0.CO;2-4     Document Type: Article

References (22)

1. Histological analysis of aggressiveness and responsiveness in Wilms tumor
2. Clonal expansion and attenuated apoptosis in Wilms tumors are associated with p53 gene mutations
3. Multivariate analysis of adverse prognostic factors in children with recurrent Wilms tumour after initial treatment according to SIOP-6 or SIOP-9 strategies
4. Prognostic factors for children with recurrent Wilms' tumor: Results from the Second and Third National Wilms' Tumor Study
5. The role of pre-operative chemotherapy in the treatment of nephroblastoma: The SIOP experience
6. Tumor volume and prognosis in patients with localized unilateral nephroblastoma treated pre-operatively
7. The effect of pre-operative therapy on the histologic features of Wilms tumor. An analysis of cases from the Third National Wilms Tumor Study
8. High telomerase reverse transcriptase (hTERT) messenger RNA level correlates with tumor recurrence in patients with favorable histology Wilms' tumor
9. Molecular genetics of relapsed Wilms' tumour analysed by comparative genomic hybridisation and WT1 and p53 Mutational Screening
10. Confirmation of FWT1 as a Wilms tumour susceptibility gene and phenotypic characteristics of Wilms tumour attributable to FWT1
11. Penetrance of mutations in familial Wilms tumor gene FWT1
12. Linkage of familial Wilms tumor predisposition to chromosome 19 and a two-locus model for the etiology of familial tumours
13. Disruption of an imprinted zinc-finger gene, ZNF215, in the Beckwith- Wiedemann syndrome
14. Risk of cancer during the first four years of life in children form the Beckwith-Wiedemann Syndrome Registry
15. The G401 cell line, utilised for studies of chromosomal changes in Wilms tumor, is derived from a rhabdoid tumor of the kidney
16. Anti-VEGF antibody suppresses primary tumor growth and metastasis in an experimental model of Wilms' tumor
17. Anti-VEGF antibody suppresses primary tumor growth and metastasis in an experimental model of Wilms' tumor
18. Truncating mutations of hSNF5/INI1 in aggressive paediatric cancer
19. Germ-line and acquired mutations of INI1 in atypical teratoid and rhabdoid tumors