Effects of angiotensin-converting enzyme inhibitor during warm blood cardioplegia

Annals of Thoracic Surgery

Volumn 70, Issue 2, 2000, Pages 627-632

  Hayashida, Nobuhiko ^a   Chihara, Shingo ^a   Tayama, Eiki ^a   Yokose, Shogo ^a   Akasu, Koji ^a   Kai, Eizo ^a   Aoyagi, Shigeaki ^a  

^a KURUME UNIVERSITY   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

CAPTOPRIL; CREATINE KINASE MB; DIPEPTIDYL CARBOXYPEPTIDASE INHIBITOR; ENDOTHELIAL LEUKOCYTE ADHESION MOLECULE 1; INTERCELLULAR ADHESION MOLECULE 1; LACTIC ACID; THIOBARBITURIC ACID REACTIVE SUBSTANCE; VASCULAR CELL ADHESION MOLECULE 1;

ANIMAL TISSUE; ARTICLE; CARDIOPLEGIA; CONTROLLED STUDY; DRUG EFFECT; ENDOTHELIUM CELL; ENZYME INHIBITION; HEART PROTECTION; ISOLATED HEART; LIPID PEROXIDATION; NONHUMAN; PRIORITY JOURNAL; RAT;

ANGIOTENSIN-CONVERTING ENZYME INHIBITORS; ANIMALS; CAPTOPRIL; HEART; HEART ARREST, INDUCED; LIPID PEROXIDATION; RATS; RATS, WISTAR; VENTRICULAR FUNCTION, LEFT; VENTRICULAR PRESSURE;

EID: 0033836313     PISSN: 00034975     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0003-4975(00)01390-4     Document Type: Article

References (27)

1. Ischemia-reperfusion injury
2. Tepid antegrade and retrograde cardioplegia
3. Impaired endothelium dependent relaxation after cardiac global ischemia and reperfusion: Role of warm blood cardioplegia
4. Effect of captopril on morbidity in patients with left ventricular dysfunction after myocardial infarction
5. Does captopril attenuate reperfusion-induced myocardial dysfunction by scavenging free radicals?
6. Ischemia-reperfusion impairs endothelium-dependent relaxation of coronary microvessels but does not affect large arteries
7. Time course of endothelial dysfunction and myocardial injury during myocardial ischemia and reperfusion in the cat
8. Coronary endothelial injury after cardiopulmonary bypass and ischemic cardioplegia is mediated by oxygen-derived free radicals
9. Effect of enalapril on myocardial infarction and unstable angina in patients with low ejection fractions
10. ISIS-4: A randomized factorial trial assessing early oral captopril, oral mononitrate, and intravenous magnesium sulphate in 58050 patients with suspected acute myocardial infarction
11. Direct scavenging of free radicals by captopril, an angiotensin converting enzyme inhibitor
12. Antioxidant effects of angiotensin-converting enzyme (ACE) inhibitors: Free radical and oxidant scavenging are sulfhydryl dependent, but lipid peroxidation is inhibited by both sulfhydryl- and nonsulfhydryl-containing ACE inhibitors
13. Angiotensin II stimulates NADH and NADPH oxidase activity in cultured vascular smooth muscle cells
14. Captopril and enalaprilat do not scavenge the superoxide anion
15. ACE-inhibition induces NO-formation in cultured bovine endothelial cells and protects isolated ischemic rat hearts
16. Inhibition of nitric oxide synthase prevents myocardial protection by ramiprilat
17. Nitric oxide, an endothelial cell relaxation factor, inhibits neutrophil superoxide anion production via a direct action on the NADPH oxidase
18. Diminished basal nitric oxide release after myocardial ischemia and reperfusion promotes neutrophil adhesion to coronary endothelium
19. Effects of supplemental L-arginine during warm blood cardioplegia
20. Induction of the adhesion molecule E-selectin in human cardiac endothelial cells by angiotensin II
21. The role of nitric oxide and cell adhesion molecules on the microcirculation in ischemia-reperfusion
22. Coronary endothelial and cardiac protective effects of a monoclonal antibody to intercellular adhesion molecule-1 in myocardial ischemia and reperfusion
23. Monoclonal antibody to intercellular adhesion molecule-1, but not to endothelial-leukocyte adhesion molecule-1, protects against myocardial ischemia/reperfusion damage in anesthetized monkeys
24. E-selectin and intercellular adhesion molecule-1 are released by activated human endothelial cells in vitro
25. Soluble forms of E-selectin, ICAM-1 and VCAM-1 are present in supernatants of cytokine-activated cultured endothelial cells
26. Inhibition of p38 mitogen-activated protein kinase decreased cardiomyocyte apoptosis and improves cardiac function after myocardial ischemia and reperfusion
27. Signaling pathways in reactive oxygen species-induced cardiomyocyte apoptosis