A neuroimmune interaction in painful peripheral neuropathy

Clinical Journal of Pain

Volumn 16, Issue 3 SUPPL., 2000, Pages

  Bennett, G J ^a  

^a DREXEL UNIVERSITY COLLEGE OF MEDICINE   (United States)

Author keywords

Allodynia; Hyperalgesia; Inflammatory response; Neuropathic pain

Indexed keywords

CYCLOSPORIN A; THALIDOMIDE;

ALLODYNIA; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CONTROLLED STUDY; DOSE RESPONSE; DRUG EFFECT; HYPERALGESIA; IMMUNE RESPONSE; MALE; NEURITIS; NEUROIMMUNOLOGY; NONHUMAN; PERIPHERAL NERVE INJURY; PERIPHERAL NEUROPATHY; PRIORITY JOURNAL; RAT; SCIATIC NERVE;

EID: 0033835826     PISSN: 07498047     EISSN: None     Source Type: Journal    
DOI: 10.1097/00002508-200009001-00005     Document Type: Article

References (9)

1. Neuropathic pain from an experimental neuritis of the rat sciatic nerve
2. Immunomodulation by thalidomide and thalidomide analogues
3. Molecular mechanisms of the immunosuppressive action of cyclosporin A
4. A peripheral mononeuropathy in rat that produces disorders of pain sensation like those seen in man
5. The local cellular response in peripheral nerves in a painful mononeuropathy results from application of 'non-constrictive' plastic cuff
6. The effect of thalidomide treatment on vascular pathology and hyperalgesia caused by chronic constriction injury of rat nerve
7. Endoneurial injection of TNF-alpha produces neuropathic pain behaviors
8. Schwann cells produce tumor necrosis factor alpha: Expression in injured and non-injured nerves
9. Tumor necrosis factor-alpha induces ectopic activity in nociceptive primary afferent fibers