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Volumn 13, Issue 10, 2000, Pages 1020-1027
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Activation of AP-1 through the MAP kinase pathway: A potential mechanism of the carcinogenic effect of arenediazonium ions
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Author keywords
[No Author keywords available]
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Indexed keywords
ACETYLCYSTEINE;
ACETYLSALICYLIC ACID;
ANTIOXIDANT;
DIAZONIUM COMPOUND;
LUCIFERASE;
MITOGEN ACTIVATED PROTEIN KINASE;
POLYCYCLIC AROMATIC HYDROCARBON DERIVATIVE;
RADICAL;
SCAVENGER;
STRESS ACTIVATED PROTEIN KINASE;
TRANSCRIPTION FACTOR AP 1;
ANIMAL CELL;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANIMAL TISSUE;
ARTICLE;
CARCINOGENICITY;
CONTROLLED STUDY;
ENZYME ACTIVATION;
ENZYME ANALYSIS;
ENZYME INDUCTION;
ENZYME INHIBITION;
ENZYME PHOSPHORYLATION;
EPITHELIUM CELL;
FEMALE;
MALE;
MOUSE;
NONHUMAN;
SIGNAL TRANSDUCTION;
TISSUE TYPES;
TRANSGENIC MOUSE;
ACETYLCYSTEINE;
ANIMALS;
CARCINOGENS;
CELL LINE;
DIAZONIUM COMPOUNDS;
ELECTRON SPIN RESONANCE SPECTROSCOPY;
ENZYME ACTIVATION;
ENZYME ACTIVATORS;
FREE RADICAL SCAVENGERS;
INDICATORS AND REAGENTS;
LUCIFERASES;
MICE;
MICE, TRANSGENIC;
MITOGEN-ACTIVATED PROTEIN KINASES;
PHOSPHORYLATION;
PLASMIDS;
PROTEIN KINASES;
TRANSCRIPTION FACTOR AP-1;
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EID: 0033794402
PISSN: 0893228X
EISSN: None
Source Type: Journal
DOI: 10.1021/tx000068s Document Type: Article |
Times cited : (21)
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References (62)
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