Response to John Cairns: The contribution of transiently hypermutable cells to mutation in stationary phase [2]

Genetics

Volumn 156, Issue 2, 2000, Pages 925-926

  Bull, Harold J ^a,b   McKenzie, Gregory J ^a   Hastings, P J ^a   Rosenberg, Susan M ^a  

^a BAYLOR COLLEGE OF MEDICINE   (United States)

^b UNIVERSITY OF SASKATCHEWAN   (Canada)

Author keywords

[No Author keywords available]

Indexed keywords

FLUCYTOSINE;

DRUG RESISTANCE; GENE ISOLATION; GENE MUTATION; LACTOSE OPERON; LETTER; MUTATION RATE; NONHUMAN; POPULATION GENETICS; POPULATION MODEL; PRIORITY JOURNAL;

EID: 0033790292     PISSN: 00166731     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Letter

References (8)

1. Evidence that stationary-phase hypermutation in the Escherichia coli chromosome is promoted by recombination
2. Appendix
3. Nonadaptive mutations occur in the F' episome during adaptive mutation conditions in Escherichia coli
4. Glycolysis
5. Spontaneous point mutations that occur more often when advantageous than when neutral
6. Recombination in adaptive mutation
7. The role of transient hypermutators in adaptive mutation in Escherichia coli
8. Genome-wide hypermutation in a subpopulation of stationary-phase cells underlies recombination-dependent adaptive mutation