Polymorphisms of the P-selectin gene and risk of myocardial infarction in men and women in the ECTIM extension study

Heart

Volumn 84, Issue 5, 2000, Pages 548-552

  Kee, F ^a   Baxter, G F ^a   Morrison, C ^b   Evans, A E ^c   McCrum, E ^c   McMaster, D ^a   Dallongeville, J ^d   Nicaud, V ^e   Poirier, O ^e   Cambien, F ^e  

^a QUEEN'S UNIVERSITY BELFAST   (United Kingdom)

^b NHS GREATER GLASGOW AND CLYDE   (United Kingdom)

^c MONICA Project   (United Kingdom)

^d INSTITUT PASTEUR DE LILLE   (France)

^e INSERM   (France)

Author keywords

Atherogenesis; Cell adhesion molecules; P selectin

Indexed keywords

CHOLESTEROL; GENE PRODUCT; PADGEM PROTEIN; PROLINE; THREONINE;

ADULT; AGE; ARTICLE; BLOOD PRESSURE; BODY MASS; CONTROLLED STUDY; DEMOGRAPHY; DNA POLYMORPHISM; FEMALE; GENE AMPLIFICATION; GENETIC SUSCEPTIBILITY; HEART INFARCTION; HUMAN; HYPERCHOLESTEROLEMIA; INTERVIEW; LIFESTYLE; MAJOR CLINICAL STUDY; MALE; POLYMERASE CHAIN REACTION; PRIORITY JOURNAL; QUESTIONNAIRE; RISK FACTOR; SEX DIFFERENCE; UNITED KINGDOM;

EID: 0033773743     PISSN: 13556037     EISSN: None     Source Type: Journal    
DOI: 10.1136/heart.84.5.548     Document Type: Article

References (12)

1. The combined role of P and E selectins in atherosclerosis
2. DNA polymorphism in adhesion molecule genesa new risk factor for early atherosclerosis
3. The P-selectin gene is highly polymorphic: Reduced frequency of the Pro715 allele carriers in patients with myocardial infarction
4. Studies on the composition and structure of plasma lipoproteins: Distribution of lipoprotein families in major density classes of normal human plasma lipoproteins
5. Simple salting out procedure for extracting DNA from human nucleated cells
6. Incidence, delay and survival in the Belfast MONICA project coronary event register
7. Remodelling and neointimal formation in the carotid artery of normal and P-selectin deficient mice
8. Soluble P-selectin is released into the coronary artery circulation after coronary spasm
9. Post angioplasty restenosis: Platelet activation and the coagulation fibrinolysis system as possible factors in the pathogenesis of restenosis
10. Circulating vascular cell adhesion molecule-I correlates with the extent of human atherosclerosis in contrast to circulating adhesion molecule I, E-selectin, P-selectin and thrombomodulin
11. Increased soluble P-selectin following myocardial infarction: A new marker for progression of atherosclerosis
12. Identification of functional site (amino acids 412-416) on the complement like repeat domains implicated in P-selectin (CD62P) leukocyte interactions