Gender differences in hemodynamic responses to endothelin-1

Journal of Cardiovascular Pharmacology

Volumn 36, Issue 5 SUPPL. 1, 2000, Pages

  Tatchum Talom, R ^a   Martel, C ^a   Labrie, C ^a   Labrie, F ^a   Marette, A ^a  

^a LAVAL UNIVERSITY   (Canada)

Author keywords

Blood pressure; Endothelin 1 (ET 1); Estrogens; Hindlimb blood flow; Sex

Indexed keywords

ENDOTHELIN 1;

ADOLESCENT; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTERIAL PRESSURE; ARTICLE; CARDIOVASCULAR RISK; CONTROLLED STUDY; FEMALE; GENDER; HEMODYNAMICS; HYPOTENSION; ISCHEMIC HEART DISEASE; MALE; NONHUMAN; PREMENOPAUSE; PRIORITY JOURNAL; VASOCONSTRICTION; VASODILATATION;

EID: 0033761947     PISSN: 01602446     EISSN: None     Source Type: Journal    
DOI: 10.1097/00005344-200036001-00066     Document Type: Article

References (10)

1. Endothelins: Molecular biology, biochemistry, pharmacology, physiology, and pathophysiology
2. Estrogen replacement therapy and coronary heart disease: Quantitative assessment of the epidemiologic evidence
3. Estrogen regulates myogenic tone in pressurized cerebral arteries by enhanced basal release of nitric oxide
4. 17β-Estradiol acutely improves endothelium-dependent relaxation to bradykinin in isolated human coronary arteries
5. Estrogen preserves regulation of shear stress by nitric oxide in arterioles of female hypertensive rats
6. Acute effect of 17β-estradiol on rabbit coronary artery contractile responses to endothelin-1
7. Effect of 17β-estradiol on coronary microvascular responses to endothelin-1
8. Endothelin-1 sensitivity of porcine coronary arteries is reduced by exercise training and is gender dependent
9. Postmenopausal hormonal replacement decreases plasma levels of endothelin-1
10. Endothelin levels decrease after oral and non oral estrogen in post-menopausal women with increased cardiovascular risk factors