Bacterial translocation and tumor necrosis factor-α gene expression in experimental hemorrhagic shock

Critical Care Medicine

Volumn 28, Issue 11, 2000, Pages 3705-3709

  Tani, Tohru ^a   Fujino, Mitsuhiro ^a   Hanasawa, Kazuyoshi ^a   Shimizu, Tomoharu ^a   Endo, Yoshihiro ^a   Kodama, Masashi ^a  

^a SHIGA UNIVERSITY OF MEDICAL SCIENCE   (Japan)

Author keywords

Cytokines; Gastrointestinal tract; Hyperoxia; Immunosuppression; In situ hybridization; Ischemia; Mesenteric lymph nodes; Messenger RNA; Paneth cell; RNA probe

Indexed keywords

CYTOKINE; MESSENGER RNA; TUMOR NECROSIS FACTOR ALPHA;

ANIMAL MODEL; ANIMAL TISSUE; ARTERIAL BLOOD; ARTICLE; BACTERIAL TRANSLOCATION; CONTROLLED STUDY; GENE EXPRESSION; HEMORRHAGIC SHOCK; HYPOXIA; IN SITU HYBRIDIZATION; MALE; MESENTERY LYMPH NODE; NONHUMAN; PRIORITY JOURNAL; RAT; SHOCK;

EID: 0033710371     PISSN: 00903493     EISSN: None     Source Type: Journal    
DOI: 10.1097/00003246-200011000-00028     Document Type: Article

References (35)

1. Translocation of certain indigenous bacteria from the gastrointestinal tract to the mesenteric lymph nodes and other organs in a gnotobiotic mouse model
2. The tumor necrosis factor ligand and receptor families
3. Another negative clinical trial of a new agent for the treatment of sepsis
4. Cachectin/tumor necrosis factor: Production, distribution and metabolic fate in vivo
5. Tumor necrosis factor alpha in cerebrospinal fluid during bacterial, but not viral, meningitis
6. Tumor necrosis factor a-producing cells in the intestinal mucosa of children with inflammatory bowel disease
7. Multiple-organ-failure syndrome
8. The gastrointestinal tract: The 'motor' of MOF
9. Splanchnic origin of cytokines in a porcine model of mesenteric ischemia-reperfusion
10. Liver as a focus of impaired oxygenation and cytokine production in a porcine model of endotoxicosis
11. Mechanism of increased susceptibility to infection following hemorrhage
12. Mucosal lesions in the small intestine of the cat during low flow
13. The intestinal mucosal lesions in shock. I. Studies on the pathogenesis
14. The intraluminal administration of perfluorochemicals to the ischemic gastrointestinal tract
15. Effect of intraluminal oxygen on experimental ischemia of the intestine
16. Effects of hyperoxia on BT and mortality during gut-derived sepsis
17. Hemorrhagic shock induces BT from the gut
18. Obstructive jaundice promotes BT from the gut
19. Overexpression and localization of cyclin D1 mRNA and antigen in esophageal cancer
20. Thermal injury promotes bacterial translocation from the gastrointestinal tract in mice with impaired T-cell-mediated immunity
21. Bacterial translocation and gram-negative bacteremia in patients with hematological malignancies
22. Endotoxemia and bacteremia during hemorrhagic shock: The link between trauma and sepsis?
23. Cadaveric small bowel and small bowel-liver transplantation in humans
24. The effect of rejection and graft-versus-host disease on small intestinal microflora and bacterial translocation after rat small bowel transplantation
25. The gut as source of sepsis after hemorrhagic shock
26. Evidence favoring the role of the gut as a cytokine-generating organ in rats subjected to hemorrhagic shock
27. Defense mechanisms in Peyer's patches and mesenteric lymph nodes against Yersinia enterocolitica involve integrins and cytokines
28. Gut ischemia and mesenteric synthesis of inflammatory cytokines after hemorrhagic or endotoxic shock
29. Gut-derived mesenteric lymph but not portal blood increases endothelial cell permeability and promotes lung injury after hemorrhagic shock
30. The biology of cachectin/TNF: A primary mediator of the host response
31. Tumor necrosis factor mRNA localized to Paneth cells of normal murine intestinal epithelium by in situ hybridization
32. A distinct array of proinflammatory cytokines is expressed in human colon epithelial cells in response to bacterial invasion
33. Occurrence of bacteremia during and after hemorrhagic shock
34. Studies of the route, magnitude, and time course of bacterial translocation in a model of systemic inflammation
35. The role of the sympathetic nervous system in the modulation of immune responses