Tumor necrosis factor-α and lipopolysaccharides induce differentially interleukin 8 and Growth Related Oncogene-α expression in human endothelial cell line EA.hy926 [7]

Annals of Plastic Surgery

Volumn 45, Issue 6, 2000, Pages 681-683

  Song, Y H ^a   Neumeister, M W ^a   Mowlavi, A ^a   Suchy, H ^a  

^a SOUTHERN ILLINOIS UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

INTERLEUKIN 8; LIPOPOLYSACCHARIDE; TUMOR NECROSIS FACTOR ALPHA;

CONTROLLED STUDY; CYTOKINE PRODUCTION; ENDOTHELIUM CELL; GENE EXPRESSION; GRAM NEGATIVE BACTERIUM; HUMAN; HUMAN CELL; INFLAMMATION; LETTER; MUSCLE ISCHEMIA; ONCOGENE; PRIORITY JOURNAL; SKELETAL MUSCLE;

EID: 0033671978     PISSN: 01487043     EISSN: None     Source Type: Journal    
DOI: 10.1097/00000637-200045060-00028     Document Type: Letter

References (10)

1. Each step during transendothelial migration of flowing neutrophils is regulated by the stimulatory concentration of tumour necrosis factor-alpha
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3. Staphylococcal enterotoxin A-induced injury of human lung endothelial cells and IL-8 accumulation are mediated by TNF-alpha
4. Release of CXC-chemokines by human lung microvascular endothelial cells (LMVEC) compared with macrovascular umbilical vein endothelial cells
5. Hypoxic induction of interleukin-8 gene expression in human endothelial cells
6. Efficient gene transfer to human endothelial cells using DNA complexed to adenovirus particles
7. Complement activation following reoxygenation of hypoxic human endothelial cells: Role of intracellular reactive oxygen species, NF-kB and new protein synthesis
8. Hypoxia/reoxygenation stimulates endothelial cells to promote interleukin-1 and interleukin-6 production. Effects of free radical scavengers
9. Tumor necrosis factor, interleukin 6, and acute phase response following hepatic ischemia/reperfusion
10. Requirements for tumor necrosis factor-alpha and interleukin-1 in limb ischemia-reperfusion injury and associated lung injury