Management of hyperinsulinism in infancy and childhood

Annals of Medicine

Volumn 32, Issue 8, 2000, Pages 544-551

  Hussain, K ^a   Aynsley Green, A ^a  

^a UNIVERSITY COLLEGE LONDON   (United Kingdom)

Author keywords

Child; Hyperinsulinism; Hypoglycaemia; Infant; Neonate

Indexed keywords

CALCIUM CHANNEL; CARBOHYDRATE; CHLOROTHIAZIDE; DIAZOXIDE; GLUCAGON; GLUCOSE; INSULIN; NIFEDIPINE; OCTREOTIDE; POTASSIUM CHANNEL; SOMATOSTATIN DERIVATIVE;

ABDOMINAL DISTENSION; ARTICLE; CARBOHYDRATE INTAKE; CHILD; CHOLELITHIASIS; CLINICAL FEATURE; DRUG EFFECT; FEEDING DISORDER; FLUID RETENTION; GENE MUTATION; GLUCOSE INFUSION; HUMAN; HYPERINSULINISM; HYPERTRICHOSIS; HYPERURICEMIA; HYPOGLYCEMIA; HYPOKALEMIA; HYPOTENSION; INFANT; LETHARGY; LEUKOPENIA; MAJOR CLINICAL STUDY; NAUSEA; NERVOUSNESS; ONSET AGE; PANCREAS RESECTION; PATHOGENESIS; PRIORITY JOURNAL; SEIZURE; SIDE EFFECT; SLOW RELEASE FORMULATION; STEATORRHEA; THROMBOCYTOPENIA; TREATMENT PLANNING; VOMITING;

EID: 0033653702     PISSN: 07853890     EISSN: None     Source Type: Journal    
DOI: 10.3109/07853890008998834     Document Type: Article

References (30)

1. Hyperinsulinism in infancy and childhood
2. Persistent hyperinsulinaemic hypoglycaemic of infancy: Therapy, clinical outcome and mutational analysis
3. Loss of functional K(ATP) channels in B-cells causes persistent hyperinsulinaemic hypoglycaemia in infancy
4. Therapy for persistent hyperinsulinaemic hypoglycaemia of infancy
5. Potassium selective ion channels in insulin secreting cells: Physiology, pharmacology and their role in stimulus secretion coupling
6. Recent advances in hyperinsulinism and the pathogenesis of diabetes mellitus
7. A point mutation inactivating the sulphonylurea receptor causes the severe form of persistent hyperinsulinaemic hypoglycaemia of infancy in Finland
8. Ionic control of the B-cell function in nesidioblastosis, a possible role for calcium channel blockade
9. Neonatal hypoglycaemia - The controversy regarding definition
10. Hypoglycaemia in infancy: The need for a rational definition
11. The practical management of hyperinsulinism in infancy
12. Genetics of neonatal hyperinsulinism
13. Hyperinsulinism (HI) in childhood associated with a defect in Short Chain 3-Hydroxyacyl CoA Dehydrogenase Deficiency (SCHAD): A new syndrome linking abnormal fatty acid metabolism and HI
14. Familial hyperinsulinism caused by activating glucokinase mutation
15. Hyperinsulinism and hyperammonaemia in infants with regulatory mutations of the glutamate dehydrogenase gene
16. Hyperinsulinism (HI) associated with sensorineural hearing loss and an inflammatory enteropathy: A new syndrome
17. Somatic deletion of the imprinted 11p15.1 region in sporadic persistent hyper-insulinaemic hypoglycaemia of infancy is specific of focal adenomatous hyperplasia and endorses partial pancreatectomy
18. The role of calcium ions in determining insulin hypersecretion in patients with PHHI
19. Glycaemic response to glucagon during fasting hypoglycaemia: An aid in the diagnosis of Hyperinsulinism
20. Neonatal hyperinsulinism (HI) is associated with abnormal cortisol responses to hypoglycaemia (HY)
21. Conservative treatment and long term follow-up of persistent hyperinsulinism of infancy (HI)
22. Partial pancreatectomy as a curative treatment in neonatal persistent hyperinsulinaemic hypoglycaemia of infancy
23. Clinical features of 52 neonates with hyperinsulinism
24. The basic structural lesion in persistent neonatal hypoglycaemia with hyperinsulinism: Deficiency of pancreatic D cells or hyperactivity of B-cells?
25. Hyperinsulinaemic hypoglycaemia of infancy (nesidioblastosis) in clinical remission: High incidence of diabetes mellitus and persistent B-cell dysfunction at long term follow up
26. Management of secondary diabetes mellitus after pancreatectomy in infancy
27. Pancreatic exocrine and endocrine function after pancreatectomy for persistent hyperinsulinaemic hypoglycaemia of infancy
28. Surface electrogastrographic evidence of forgut dysmotility in persistent hyperinsulinaemic hypoglycaemia of infancy (PHHI)
29. Engineering a glucose-responsive human insulin-secreting cell line from islets of Langerhans from a patient with persistent hyperinsulinaemic hypoglycaemia of infancy (PHHI)
30. Nesidioblastosis unravelled