Placental vascularisation requires the AP-1 component Fra1

Development

Volumn 127, Issue 22, 2000, Pages 4937-4948

  Schreiber, M ^a   Wang, Z Q ^a   Jochum, W ^a   Fetka, I ^a   Elliott, C ^a   Wagner, E F ^a  

^a RESEARCH INSTITUTE OF MOLECULAR PATHOLOGY   (Austria)

Author keywords

AP 1; Embryonic lethality; Fra1; Gene targeting; Mouse; Tetraploid blastocyst injection

Indexed keywords

IMMEDIATE EARLY PROTEIN; TRANSCRIPTION FACTOR AP 1;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BLASTOCYST; CELL COUNT; CONTROLLED STUDY; EMBRYO CELL; ENDOTHELIUM CELL; FETUS DEATH; GENE EXPRESSION; GENE FUNCTION; GENE INACTIVATION; GENE LOSS; GENE TARGETING; IMMEDIATE EARLY GENE; INTRAUTERINE GROWTH RETARDATION; LETHALITY; MARKER GENE; MOUSE; MUTANT; NONHUMAN; PERINATAL PERIOD; PLACENTA CIRCULATION; PRIORITY JOURNAL; STEM CELL; SURVIVAL; TROPHOBLAST; VASCULAR ENDOTHELIUM; YOLK SAC;

ANIMALS; BASE SEQUENCE; CELL DIVISION; DNA PRIMERS; FEMALE; FETAL DEATH; FETAL GROWTH RETARDATION; GENES, IMMEDIATE-EARLY; GESTATIONAL AGE; MALE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; NEOVASCULARIZATION, PHYSIOLOGIC; PLACENTA; PREGNANCY; PROTO-ONCOGENE PROTEINS C-FOS; TRANSCRIPTION FACTOR AP-1; YOLK SAC;

EID: 0033638817     PISSN: 09501991     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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