Increased inflammation and intracellular calcium caused by ultrafine carbon black is independent of transition metals or other soluble components

Occupational and Environmental Medicine

Volumn 57, Issue 10, 2000, Pages 685-691

  Brown, D M ^a   Stone, V ^a   Findlay, P ^a   MacNee, W ^a   Donaldson, K ^a  

^a EDINBURGH NAPIER UNIVERSITY   (United Kingdom)

Author keywords

Calcium; Transition metals; Ultrafine

Indexed keywords

CALCIUM; CARBON; CITRIC ACID; DEFEROXAMINE MESYLATE; IRON; METAL; METAL CHELATE;

AIR POLLUTION; ANIMAL EXPERIMENT; ARTICLE; CALCIUM CELL LEVEL; CALCIUM HOMEOSTASIS; CONTROLLED STUDY; FEMALE; HUMAN; HUMAN CELL; IN VITRO STUDY; IN VIVO STUDY; LUNG ALVEOLUS MACROPHAGE; LUNG LAVAGE; NONHUMAN; PARTICULATE MATTER; PRIORITY JOURNAL; RAT; RESPIRATORY TRACT INFLAMMATION; SURFACE PROPERTY;

AIR POLLUTANTS; ANIMALS; BRONCHOALVEOLAR LAVAGE FLUID; CALCIUM; CARBON; CHELATING AGENTS; DEFEROXAMINE; DUST; FEMALE; INFLAMMATION; IRON; MACROPHAGES, ALVEOLAR; PARTICLE SIZE; RATS; RATS, WISTAR;

EID: 0033626747     PISSN: 13510711     EISSN: None     Source Type: Journal    
DOI: 10.1136/oem.57.10.685     Document Type: Article

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