Nature and nurture in systemic lupus erythematosus

Advances in Experimental Medicine and Biology

Volumn 455, Issue , 1999, Pages 7-13

  Maddison, P J ^a  

^a YSBYTY GWYNEDD   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

ANTIGEN EXPRESSION; ANTIGEN SPECIFICITY; APOPTOSIS; CONFERENCE PAPER; ENVIRONMENTAL FACTOR; GENETIC SUSCEPTIBILITY; HUMAN; IMMUNE RESPONSE; IMMUNOLOGICAL TOLERANCE; MAJOR HISTOCOMPATIBILITY COMPLEX; PRIORITY JOURNAL; REPORTER GENE; SYSTEMIC LUPUS ERYTHEMATOSUS;

EID: 0033282340     PISSN: 00652598     EISSN: None     Source Type: Book Series    
DOI: 10.1007/978-1-4615-4857-7_2     Document Type: Conference Paper

References (17)

1. FcgammaRIIA alleles are heritable risk factors for lupus nephritis
2. FcγIIa polymorphism in Caucasian SLE patients: Association with clinical symptoms
3. A novel polymorphism of FcγIIIA, which alters function, associated with the SLE phenotype
4. Evidence for linkage of a candidate chromosome 1 region to human systemic lupus erythematosus
5. A susceptibility gene for SLE maps to a 5cM region of chromosome 1q
6. Drug-induced lupus
7. The cutaneous signs of lupus erythematosus
8. Serologic subsets in systemic lupus erythematosus: An examination of autoantibodies in relationship to clinical features of disease and HLA antigens
9. The genetics of human lupus
10. Systemic lupus erythematosus and genes within the HLA region
11. Heritable major histocompatibility complex class-II associated differences in production of tumor necrosis factor alpha: Relevance to genetic predisposition to systemic lupus erythematosus
12. Autoantigenicity of Ro/SSA antigen is related to a nucleocapsid protein of vesicular stomatitis virus
13. Ultraviolet light modulation of autoantigens, epidermal cytokines and adhesion molecules as contributing factors of the pathogenesis of cutaneous LE
14. Ultraviolet light induces binding of antibodies to selected nuclear antigens on cultured human keratinocytes
15. Enhanced membrane binding of autoantibodies to cultured keratinocytes of systemic lupus erythematosus patients after ultraviolet B/ultraviolet A radiation
16. Ultraviolet light-induced keratinocyte apoptosis: A potential mechanism for the induction of skin lesions and autoantibody production in LE
17. Apoptosis in the rheumatic diseases