High fructose-fed rats: A model of glomerulosclerosis involving the renin-angiotensin system and renal gelatinases

Annals of the New York Academy of Sciences

Volumn 878, Issue , 1999, Pages 716-719

  Zaoui, Philippe ^a,b   Rossini, E ^a   Pinel, N ^a   Cordonnier, D ^a   Halimi, S ^a   Morel, F ^a  

^a UNIV GRENOBLE ALPES   (France)

^b GRENOBLE UNIVERSITY HOSPITAL   (France)

Author keywords

[No Author keywords available]

Indexed keywords

ALBUMIN; FRUCTOSE; GELATINASE; GELATINASE A; GELATINASE B; KIDNEY ENZYME; METFORMIN;

ANIMAL EXPERIMENT; ANIMAL MODEL; CONFERENCE PAPER; CONTROLLED STUDY; GLOMERULOSCLEROSIS; GLUCOSE INTOLERANCE; HYPERTRIGLYCERIDEMIA; NONHUMAN; PROTEINURIA; RAT; RENIN ANGIOTENSIN ALDOSTERONE SYSTEM;

ANIMALIA;

EID: 0032837307     PISSN: 00778923     EISSN: None     Source Type: Book Series    
DOI: 10.1111/j.1749-6632.1999.tb07771.x     Document Type: Conference Paper

References (6)

1. 1. Anderson, S.S., Y. Kim & E.C. Tsilibary. 1994. Effects of matrix glycation on mesangial cell adhesion, spreading and proliferation. Kidney Int. 46: 1359-1367.
2. 2. Ikeda, T. & T. Hoshino. 1996. An inhibition of urinary albumin excretion by protease inhibitor in streptozotocin-diabetic rats. Nephron 74: 709-712.
3. 3. Baylis, C. 1994. Age-dependent glomerular damage in the rat. Dissociation between glomerular injury and both glomerular hypertension and hypertrophy. Male gender as a primary risk factor. J. Clin. Invest. 94: 1823-1829.
4. 4. Adler, S. 1994. Structure-function relationships associated with extracellular matrix alterations in diabetic glomerulopathy. J. Am. Soc. Nephrol. 5: 1165-1172.
5. 5. Anderson, P.M., X.Y. Zhang, J. Tian, J.D. Correale, X.P. Xi, D. Yang, K. Graf, R.E. Law & W.A. Hsueh. 1996. Insulin and angiotensin II are additive in stimulating TGF-β1 and matrix mRNAs in mesangial cells. Kidney Int. 50: 745-753.
6. 6. Doi, T., L.J. Striker, C. Quaife, F.G. Conti, R. Palmiter, R. Behringer, R. Brinster & G.E. Striker. 1988. Progressive glomerulosclerosis develops in transgenic mice chronically expressing growth hormone and growth hormone releasing factor but not in those expressing insulin-like growth factor-1. Am. J. Pathol. 131: 398-403.