Toxic shock syndrome toxin-1 contributes to the arthritogenicity of Staphylococcus aureus
1 Abdelnour A, Bremell T, Tarkowski A. Toxic shock syndrome toxin-1 contributes to the arthritogenicity of Staphylococcus aureus. J Infect Dis 1994; 170:84-89.
Mutants of staphylococcal toxic shock syndrome toxin 1: Mitogenicity and recognition by a neutralizing monoclonal antibody
2 Blanco L, Choi M, Conolly K et al. Mutants of staphylococcal toxic shock syndrome toxin 1: mitogenicity and recognition by a neutralizing monoclonal antibody. Infect Immun 1990; 58:3020-8.
Histopathological and serological progression of Staphylococcus aureus arthritis
5 Bremell T, Abdelnour A, Tarkowski A. Histopathological and serological progression of Staphylococcus aureus arthritis. Infect Immun 1992; 60:2976-85.
Clonal expansion of T lymphocytes causes arthritis and mortality in mice infected with toxic shock syndrome toxin-1-producing staphylococci
6 Abdelnour A, Bremell T, Holmdahl R et al. Clonal expansion of T lymphocytes causes arthritis and mortality in mice infected with toxic shock syndrome toxin-1-producing staphylococci. Eur J Immunol 1994; 24:1161-6.
Delineation by use of specific monoclonal antibodies of the T-cell receptor and major histocompatibility complex interaction sites on the superantigen toxic shock syndrome toxin 1
8 Shimonkevitz R, Boen E, Malmstrom S et al. Delineation by use of specific monoclonal antibodies of the T-cell receptor and major histocompatibility complex interaction sites on the superantigen toxic shock syndrome toxin 1. Infect Immun 1996; 64:1133-9.
9 Kum WWS, Laupland KB, Chow AW. Defining a novel domain of staphylococcal toxic shock syndrome toxin-1 critical for major histocompatibility complex class II binding, superantigenic activity, cytokine secretion and lethality. In press.
The accessory gene regulator (agr) controls Staphylococcus aureus virulence in a murine arthritis model
10 Abdelnour A, Arvidson S, Bremell T et al. The accessory gene regulator (agr) controls Staphylococcus aureus virulence in a murine arthritis model. Infect Immun 1993; 61:3879-85.
Demonstration of immunoreactive sites on cartilage after in vivo administration of biotinylated antitype II collagen antibodies
11 Jonsson R, Karlsson AL, Holmdahl R. Demonstration of immunoreactive sites on cartilage after in vivo administration of biotinylated antitype II collagen antibodies. J Histochem Cytochem 1989; 37:265-8.
Multiple epitopes on cartilage type II collagen are accessible for antibody binding in vivo
12 Holmdahl R, Mo JA, Jonsson R et al. Multiple epitopes on cartilage type II collagen are accessible for antibody binding in vivo. Autoimmunity 1991; 10:27-34.
Arthritis and sepsis caused by Staphylococcus aureus: Can the tissue injury be reduced by modulating the host's immune system?
13 Tarkowski A, Wagner H, Arthritis and sepsis caused by Staphylococcus aureus: can the tissue injury be reduced by modulating the host's immune system? Mol Med Today 1998; 4:15-18.
Differential effects of staphylococcal toxic shock syndrome toxin-1 on B cell apoptosis
14 Newell MK, Hofer RD, Duke R et al. Differential effects of staphylococcal toxic shock syndrome toxin-1 on B cell apoptosis. Proc Natl Acad Sci USA 1996; 93:5425-30.
Mice with the xid B-cell defect are less susceptible to developing Staphylococcus aureus induced arthritis
15 Zhao Y, Abdelnour A, Holmdahl R et al. Mice with the xid B-cell defect are less susceptible to developing Staphylococcus aureus induced arthritis. J Imnumol 1995; 155:2067-76.