Bystander activation of CD4+ T cells can represent an exclusive means of immunopathology in a virus infection

European Journal of Immunology

Volumn 29, Issue 11, 1999, Pages 3674-3682

  Gangappa, Shivaprakash ^a,c   Deshpande, Shilpa P ^a   Rouse, Barry T ^a,b  

^a UNIVERSITY OF TENNESSEE   (United States)

^b UNIVERSITY OF TENNESSEE   (United States)

^c WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Bystander activation; Immunopathology; Stromal keratitis; Transgenic mouse

Indexed keywords

CD4 ANTIGEN; CD8 ANTIGEN; OVALBUMIN; VIRUS ANTIBODY; VIRUS ANTIGEN;

ADOPTIVE TRANSFER; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CONTROLLED STUDY; FEMALE; HERPES SIMPLEX KERATITIS; HERPES SIMPLEX VIRUS; IMMUNOPATHOLOGY; INFLAMMATION; MALE; MATING; MOUSE; NONHUMAN; PRIORITY JOURNAL; T LYMPHOCYTE ACTIVATION; TRANSGENIC MOUSE; VIRUS INFECTION;

ADOPTIVE TRANSFER; ANIMALS; ANTIBODIES, VIRAL; CD4-POSITIVE T-LYMPHOCYTES; CD8-POSITIVE T-LYMPHOCYTES; ENCEPHALITIS, VIRAL; FEMALE; HERPESVIRUS 1, HUMAN; IMMUNIZATION, PASSIVE; KERATITIS, HERPETIC; MALE; MICE; MICE, INBRED BALB C; MICE, SCID; MICE, TRANSGENIC;

EID: 0032703166     PISSN: 00142980     EISSN: None     Source Type: Journal    
DOI: 10.1002/(SICI)1521-4141(199911)29:11<3674::AID-IMMU3674>3.0.CO;2-7     Document Type: Article

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