H-ras activation is an early event in the ptaquiloside-induced carcinogenesis: Comparison of acute and chronic toxicity in rats

Biochemical and Biophysical Research Communications

Volumn 250, Issue 2, 1998, Pages 491-497

  Shahin, Mahmood ^a   Moore, Michael R ^a   Worrall, Simon ^c   Smith, Barry L ^b   Seawright, Alan A ^a   Prakash, Arungundrum S ^a  

^a UNIVERSITY OF QUEENSLAND   (Australia)

^b RUAKURA RESEARCH CENTRE   (New Zealand)

^c NONE

Author keywords

Adenocarcinoma; Bracken fern; DNA adduct; H ras; p53; Ptaquiloside; TNF

Indexed keywords

PTAQUILOSIDE;

ACUTE TOXICITY; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BLADDER CARCINOGENESIS; BREAST ADENOCARCINOMA; CHEMICAL CARCINOGENESIS; CHRONIC TOXICITY; COMPARATIVE STUDY; CONTROLLED STUDY; DNA ADDUCT; ESOPHAGUS CANCER; FEMALE; FERN; GENE ACTIVATION; HEMATOPOIETIC STEM CELL; KIDNEY TUBULE NECROSIS; NONHUMAN; ONCOGENE H RAS; PRIORITY JOURNAL; RAT; TUMOR SUPPRESSOR GENE;

ANIMALIA; FILICOPHYTA; PTERIDIUM; PTERIDIUM AQUILINUM;

EID: 0032544681     PISSN: 0006291X     EISSN: None     Source Type: Journal    
DOI: 10.1006/bbrc.1998.9341     Document Type: Article

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