Contribution of K(ca) channel activation to hypoxic cerebrovasodilation does not involve NO

Brain Research

Volumn 799, Issue 1, 1998, Pages 44-48

  Armstead, William M ^a  

^a UNIVERSITY OF PENNSYLVANIA   (United States)

Author keywords

Cerebral circulation; Cyclic nucleotide; Newborn

Indexed keywords

POTASSIUM CHANNEL; 1 (2 HYDROXY 5 TRIFLUOROMETHYLPHENYL) 5 TRIFLUOROMETHYL 2(3H) BENZIMIDAZOLONE; BENZIMIDAZOLE DERIVATIVE; CALCIUM ACTIVATED POTASSIUM CHANNEL; CYCLIC GMP; ENDORPHIN; ENZYME INHIBITOR; INTERMEDIATE CONDUCTANCE CALCIUM ACTIVATED POTASSIUM CHANNEL; N(G) NITROARGININE; NITRIC OXIDE; POTASSIUM CHANNEL BLOCKING AGENT;

ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN CIRCULATION; BRAIN HYPOXIA; CONTROLLED STUDY; FEMALE; MALE; NONHUMAN; PIA ARTERY; PRIORITY JOURNAL; SWINE; VASODILATATION; ANIMAL; ANOXIA; ARTERY; DRUG EFFECT; DRUG POTENTIATION; METABOLISM; PATHOPHYSIOLOGY; PHYSIOLOGY; PIA MATER; VASCULARIZATION;

ANIMALS; ANOXIA; ARTERIES; BENZIMIDAZOLES; CEREBROVASCULAR CIRCULATION; CYCLIC GMP; ENDORPHINS; ENZYME INHIBITORS; FEMALE; INTERMEDIATE-CONDUCTANCE CALCIUM-ACTIVATED POTASSIUM CHANNELS; MALE; NITRIC OXIDE; NITROARGININE; PIA MATER; POTASSIUM CHANNEL BLOCKERS; POTASSIUM CHANNELS; POTASSIUM CHANNELS, CALCIUM-ACTIVATED; SWINE; VASODILATION;

EID: 0032513987     PISSN: 00068993     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0006-8993(98)00462-4     Document Type: Article

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