Energetic basis of diastolic dysfunction

Magnetic Resonance Materials in Physics, Biology, and Medicine

Volumn 6, Issue 2-3, 1998, Pages 129-131

  Tian, Rong ^a  

^a BRIGHAM AND WOMEN S HOSPITAL   (United States)

Author keywords

Cross bridge cycling; Diastolic dysfunction; Muscle

Indexed keywords

ACTIN MYOSIN INTERACTION; CONFERENCE PAPER; DIASTOLE; ENERGY METABOLISM; ENZYME ACTIVITY; ENZYME SUBSTRATE; HEART DISEASE; HEART MUSCLE CELL; HEART MUSCLE CONDUCTION SYSTEM; HIGH PERFORMANCE LIQUID CHROMATOGRAPHY; PRIORITY JOURNAL;

ADENOSINE DIPHOSPHATE; ADENOSINE TRIPHOSPHATE; ANIMALS; CREATINE KINASE; DIASTOLE; ENERGY METABOLISM; HYPERTROPHY, LEFT VENTRICULAR; MYOCARDIUM; NUCLEAR MAGNETIC RESONANCE, BIOMOLECULAR; RATS; STROKE VOLUME; VENTRICULAR DYSFUNCTION, LEFT;

EID: 0032169720     PISSN: 13528661     EISSN: None     Source Type: Journal    
DOI: 10.1016/S1352-8661(98)00039-8     Document Type: Conference Paper

References (3)

1. Tian R, Christe ME, Spindler M et al. Role of MgADP in the development of diastolic dysfunction in the intact beating rat heart. J Clin Invest. 99:1997;745-751.
2. Kagaya Y, Weinberg EO, Ito N, Mochizuki T, Barry WH, Lorell BH Glycolytic inhibition: effects on diastolic relaxation and intracellular calcium handling in hypertrophied rat ventricular myocytes. J Clin Invest. 95:1995;2766-2776.
3. Tian R, Nascimben L, Ingwall JS, Lorell BH Failure to maintain a low ADP concentration impairs diastolic function in hypertrophied rat hearts. Circulation. 96:1997;1313-1319.