Chromosome 3p14 alterations in lung cancer: Evidence that FHIT exon deletion is a target of tobacco carcinogens and asbestos

Cancer Research

Volumn 58, Issue 9, 1998, Pages 1804-1807

  Nelson, Heather H ^a   Wiencke, John K ^b   Gunn, Laura ^a   Wain, John C ^c   Christiani, David C ^a,c   Kelsey, Karl T ^a  

^a HARVARD SCHOOL OF PUBLIC HEALTH   (United States)

^b UNIVERSITY OF CALIFORNIA   (United States)

^c MASSACHUSETTS GENERAL HOSPITAL   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ASBESTOS; CARCINOGEN;

ADULT; AGED; ARTICLE; CARCINOGENESIS; CHROMOSOME 3P; CLINICAL ARTICLE; DISEASE ASSOCIATION; EXON; FEMALE; GENE DELETION; HUMAN; HUMAN CELL; LUNG CANCER; MALE; MARKER GENE; OUTCOMES RESEARCH; POINT MUTATION; POLYMERASE CHAIN REACTION; PRIORITY JOURNAL; SINGLE STRAND CONFORMATION POLYMORPHISM; SMOKING; TOBACCO;

ACID ANHYDRIDE HYDROLASES; AGED; ASBESTOS; CARCINOMA, NON-SMALL-CELL LUNG; CHROMOSOMES, HUMAN, PAIR 3; DNA ADDUCTS; DNA PRIMERS; EXONS; FEMALE; HUMANS; LOSS OF HETEROZYGOSITY; LUNG NEOPLASMS; MALE; MIDDLE AGED; NEOPLASM PROTEINS; POINT MUTATION; POLYMERASE CHAIN REACTION; POLYMORPHISM, SINGLE-STRANDED CONFORMATIONAL; PROTEINS; SEQUENCE DELETION; SMOKING;

EID: 0032080899     PISSN: 00085472     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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