-
1
-
-
0019179123
-
Septic autocannibalism: A failure of exogenous nutritional support
-
Cerra FB, Siegel JH, Coleman B, Border JR, McMenamy RR. Septic autocannibalism: a failure of exogenous nutritional support. Ann Surg 1980; 192:570-580.
-
(1980)
Ann Surg
, vol.192
, pp. 570-580
-
-
Cerra, F.B.1
Siegel, J.H.2
Coleman, B.3
Border, J.R.4
McMenamy, R.R.5
-
2
-
-
0029879502
-
Muscle protein synthesis decreases 24 hours after abdominal surgey irrespective of total parenteral nutrition
-
Tjoder I, Essen P, Therne A, Garlick P, Wernerman J, McNurlan MA. Muscle protein synthesis decreases 24 hours after abdominal surgey irrespective of total parenteral nutrition. J Parent Enteral Nutr 1996; 20:135-138.
-
(1996)
J Parent Enteral Nutr
, vol.20
, pp. 135-138
-
-
Tjoder, I.1
Essen, P.2
Therne, A.3
Garlick, P.4
Wernerman, J.5
McNurlan, M.A.6
-
3
-
-
0026417229
-
Catabolic illness, strategies for enhancing recovery
-
Wilmore DW. Catabolic illness, strategies for enhancing recovery. N Engl J Med 1991; 325:695-702.
-
(1991)
N Engl J Med
, vol.325
, pp. 695-702
-
-
Wilmore, D.W.1
-
4
-
-
0028935349
-
Early enteral administration of a formula (Impact) supplemented with arginine, nucleotides, and fish oil in intensive care unit patients: Results of a multicenter, prospective, randomized, clinical trial
-
Bower RH, Cerra FB, Bershadsky B, Licari JJ, Hoyt DB, Jensen GJ, et al. Early enteral administration of a formula (Impact) supplemented with arginine, nucleotides, and fish oil in intensive care unit patients: results of a multicenter, prospective, randomized, clinical trial. Crit Care Med 1995; 23:436-449.
-
(1995)
Crit Care Med
, vol.23
, pp. 436-449
-
-
Bower, R.H.1
Cerra, F.B.2
Bershadsky, B.3
Licari, J.J.4
Hoyt, D.B.5
Jensen, G.J.6
-
5
-
-
0028109903
-
Clinical benefits of an immune-enhancing diet for early postinjury enteral feeding
-
Moore FA, Moore EE, Kudsk KA, Brown RO, Bower RH, Koruda MJ, et al. Clinical benefits of an immune-enhancing diet for early postinjury enteral feeding. J Trauma 1994; 37:607-615.
-
(1994)
J Trauma
, vol.37
, pp. 607-615
-
-
Moore, F.A.1
Moore, E.E.2
Kudsk, K.A.3
Brown, R.O.4
Bower, R.H.5
Koruda, M.J.6
-
6
-
-
0030638323
-
Regulation of skeletal muscle protein turnover during sepsis: Mechanisms and mediators
-
Vary TC, Kimball SR, Cooney RN. Regulation of skeletal muscle protein turnover during sepsis: mechanisms and mediators. Shock 1997; 7:1-16.
-
(1997)
Shock
, vol.7
, pp. 1-16
-
-
Vary, T.C.1
Kimball, S.R.2
Cooney, R.N.3
-
7
-
-
0030951073
-
Intracellular regulation of protein degradation during sepsis is different in fast- and slow-twitch muscle
-
Tiao G, Lieberman M, Fischer JE, Hasseigren P-O. Intracellular regulation of protein degradation during sepsis is different in fast- and slow-twitch muscle. Am J Physiol 1997; 272:R849-R856.
-
(1997)
Am J Physiol
, vol.272
-
-
Tiao, G.1
Lieberman, M.2
Fischer, J.E.3
Hasseigren, P.-O.4
-
8
-
-
0030185450
-
Sepsis inhibits synthesis of myofibrillar and sarcoplasmic proteins: Modulation by interleukin-1 receptor anatgonist
-
Vary TC, Owens EL, Beers JK, Verner K, Cooney RN. Sepsis inhibits synthesis of myofibrillar and sarcoplasmic proteins: modulation by interleukin-1 receptor anatgonist. Shock 1996; 6:13-18.
-
(1996)
Shock
, vol.6
, pp. 13-18
-
-
Vary, T.C.1
Owens, E.L.2
Beers, J.K.3
Verner, K.4
Cooney, R.N.5
-
9
-
-
0024386017
-
Total and myofibrillar protein breakdown in different types of rat skeletal muscle: Effects of sepsis and regulation by insulin
-
Hasseigren PO, James JH, Benson DW, Angeras U, Hiyama DT, Shujun LI, Fisher JE. Total and myofibrillar protein breakdown in different types of rat skeletal muscle: effects of sepsis and regulation by insulin. Metabolism 1989; 38:634-640.
-
(1989)
Metabolism
, vol.38
, pp. 634-640
-
-
Hasseigren, P.O.1
James, J.H.2
Benson, D.W.3
Angeras, U.4
Hiyama, D.T.5
Shujun, L.I.6
Fisher, J.E.7
-
10
-
-
0030924558
-
Transient exposure of human myoblasts to tumor necrosis factor-α inhibits serum and insulin-like growth factor-1 stimulated protein synthesis
-
Frost RA, Lang CH, Gelato MC. Transient exposure of human myoblasts to tumor necrosis factor-α inhibits serum and insulin-like growth factor-1 stimulated protein synthesis. Endocrinology 1997; 138:4153-4159. Addition of TNF was shown to modulate protein synthesis in myoblasts in culture. The effects of TNF were observed over a relatively long period, indicating that TNF may have effects that last beyond the period where their concentration is increased.
-
(1997)
Endocrinology
, vol.138
, pp. 4153-4159
-
-
Frost, R.A.1
Lang, C.H.2
Gelato, M.C.3
-
11
-
-
0029933665
-
Tumor necrosis factor (TNF)-α inhibits insulin signaling through stimulation of the p55TNF receptor and activation of sphingomyelinase
-
Peraldi P, Hotamisligil GS, Buurmant WA, White MF, Spiegelman BM. Tumor necrosis factor (TNF)-α inhibits insulin signaling through stimulation of the p55TNF receptor and activation of sphingomyelinase. J Biol Chem 1996; 271:13018-13022.
-
(1996)
J Biol Chem
, vol.271
, pp. 13018-13022
-
-
Peraldi, P.1
Hotamisligil, G.S.2
Buurmant, W.A.3
White, M.F.4
Spiegelman, B.M.5
-
12
-
-
0030293408
-
Herman Lecture. Relation of metabolic studies to clinical nutrition - The example of burn injury
-
Wolfe RR. Herman Lecture. Relation of metabolic studies to clinical nutrition - the example of burn injury. Am J Clin Nutr 1996; 64:800-808.
-
(1996)
Am J Clin Nutr
, vol.64
, pp. 800-808
-
-
Wolfe, R.R.1
-
14
-
-
3042901760
-
Reduced 40S initiation complex formation in skeletal muscle during sepsis
-
in press
-
Vary TC, Jurasinski C, Kimball SR. Reduced 40S initiation complex formation in skeletal muscle during sepsis. Mol Cell Biochem 1997 (in press). By examining the incorporation of [35S]methionine into 43S preinitiation complexes as isolated on CsCI density grardients, a direct effect of sepsis on one of the early steps in peptide-chain initiation was demonstrated. These studies therefore identify one biochemical locus responsible the inhibition of protein synthesis in sepsis.
-
(1997)
Mol Cell Biochem
-
-
Vary, T.C.1
Jurasinski, C.2
Kimball, S.R.3
-
15
-
-
0026760562
-
Age-dependent decrease in the amount of eukaryotic initiation factor 2 in various rat tissues
-
Kimball S, Vary TC, Jefferson LS. Age-dependent decrease in the amount of eukaryotic initiation factor 2 in various rat tissues. Biochem J 1992; 286:263-268.
-
(1992)
Biochem J
, vol.286
, pp. 263-268
-
-
Kimball, S.1
Vary, T.C.2
Jefferson, L.S.3
-
17
-
-
0030041975
-
Altered expression of eukaryotic initiation factor 2B in skeletal muscle during sepsis
-
Voisin L, Gray K, Jurasinski CV, Flowers KM, Kimball SR, Jefferson LS, Vary TC. Altered expression of eukaryotic initiation factor 2B in skeletal muscle during sepsis. Am J Physiol 1996; 270:E43-E50. These studies showed that sepsis, but not sterile inflammtion cause expression of elF-2B protein to be diminished. The decrease in protein paralleled a similar reduction in the mRNA suggesting that the protein content is regulated by mRNA expression. No alteration in elF-2B activity was observed in heart, a tissue where protein synthesis is not affected by sepsis. The depletion of elF-2B protein provides in part a biochemical reason for reduced elF-2B activity in fast-twitch muscle during sepsis.
-
(1996)
Am J Physiol
, vol.270
-
-
Voisin, L.1
Gray, K.2
Jurasinski, C.V.3
Flowers, K.M.4
Kimball, S.R.5
Jefferson, L.S.6
Vary, T.C.7
-
18
-
-
0029819257
-
Regulation of peptide-chain initiation during sepsis by interleukin-1 receptor antagonist
-
Vary TC, Voisin L, Cooney RN. Regulation of peptide-chain initiation during sepsis by interleukin-1 receptor antagonist. Am J Physiol 1996; 271:E513-E520. By using a specific inhibitor of IL-1, the authors were able to demonstrate that prevention of the fall in protein synthesis with this compound was attributed to a maintanence of elF-2B protein and mRNA at levels observed in control individuals.
-
(1996)
Am J Physiol
, vol.271
-
-
Vary, T.C.1
Voisin, L.2
Cooney, R.N.3
-
19
-
-
3042972367
-
Amrinone prevents muscle protein wasting during chronic sepsis
-
Juransinski CV, Kilpatrick L, Vary TC. Amrinone prevents muscle protein wasting during chronic sepsis. Am J Physiol 1994; 268:E636-E641.
-
(1994)
Am J Physiol
, vol.268
-
-
Juransinski, C.V.1
Kilpatrick, L.2
Vary, T.C.3
-
20
-
-
0027130313
-
Pentoxyfylline decreases the body weight loss and muscle protein wasting characteristic of sepsis
-
Breuille D, Farge MC, Rose R, Arnal M, Obled C. Pentoxyfylline decreases the body weight loss and muscle protein wasting characteristic of sepsis. Am J Physiol 1993; 265:E660-E666.
-
(1993)
Am J Physiol
, vol.265
-
-
Breuille, D.1
Farge, M.C.2
Rose, R.3
Arnal, M.4
Obled, C.5
-
21
-
-
0030593939
-
Mechanism of muscle wasting. Role of ubiqutin-proteasome pathway
-
Mitch WE, Goldberg AL. Mechanism of muscle wasting. Role of ubiqutin-proteasome pathway. N Engl J Med 1996; 335:1897-1905.
-
(1996)
N Engl J Med
, vol.335
, pp. 1897-1905
-
-
Mitch, W.E.1
Goldberg, A.L.2
-
22
-
-
0030897196
-
The ubiquitin-proteasome pathway: A review of a novel intracellular mechanism of muscle protein breakdown during sepsis and other catabolic conditions
-
Hasselgren P-O, Fisher JE. The ubiquitin-proteasome pathway: a review of a novel intracellular mechanism of muscle protein breakdown during sepsis and other catabolic conditions. Ann Surg 1997; 225:307-316.
-
(1997)
Ann Surg
, vol.225
, pp. 307-316
-
-
Hasselgren, P.-O.1
Fisher, J.E.2
-
23
-
-
0342592320
-
The ubiquitin-dependent proteolytic pathway in skeletal muscle: Its role in pathological states
-
Argilles JM, Lopez-Soriano FJ. The ubiquitin-dependent proteolytic pathway in skeletal muscle: its role in pathological states. Trends Pharmacol Sci 1996; 17:223-226.
-
(1996)
Trends Pharmacol Sci
, vol.17
, pp. 223-226
-
-
Argilles, J.M.1
Lopez-Soriano, F.J.2
-
24
-
-
0027973469
-
Sepsis stimulates non-lysosomal, energy-dependent proteolysis and increases ubiquitin mRNA levels in skeletal muscle
-
Tiao G, Fagan JM, Samuels N, James JH, Hudson K, Lieberman M, et al. Sepsis stimulates non-lysosomal, energy-dependent proteolysis and increases ubiquitin mRNA levels in skeletal muscle. J Clin Invest 1994; 94:2255-2264.
-
(1994)
J Clin Invest
, vol.94
, pp. 2255-2264
-
-
Tiao, G.1
Fagan, J.M.2
Samuels, N.3
James, J.H.4
Hudson, K.5
Lieberman, M.6
-
25
-
-
0041173139
-
Sepsis increases proteasome proteolysis and mRNA levels for proteasome subunit RC3 in skeletal muscle
-
Tiao G, Fagan JM, Lieberman M, Fischer JE, Hasselgren P-O. Sepsis increases proteasome proteolysis and mRNA levels for proteasome subunit RC3 in skeletal muscle. Surg Forum 1995; 46:10-12.
-
(1995)
Surg Forum
, vol.46
, pp. 10-12
-
-
Tiao, G.1
Fagan, J.M.2
Lieberman, M.3
Fischer, J.E.4
Hasselgren, P.-O.5
-
26
-
-
0031018516
-
Sepsis is associated with increased mRNAs of the ubiquitin-proteasome proteolytic pathway in human skeletal muscle
-
Tiao G, Wang JJ, Meyer TA, Luchette FA, Fischer JE, Hasselgren P-O. Sepsis is associated with increased mRNAs of the ubiquitin-proteasome proteolytic pathway in human skeletal muscle. J Clin Invest 1997; 99:163-168. These observation suggest that the changes observed in the ubiquitin-proteasome pathway in animal models are also present in skeletal muscle from human septic patients.
-
(1997)
J Clin Invest
, vol.99
, pp. 163-168
-
-
Tiao, G.1
Wang, J.J.2
Meyer, T.A.3
Luchette, F.A.4
Fischer, J.E.5
Hasselgren, P.-O.6
-
27
-
-
0029883726
-
2+-activated, and ubiquitin-proteasome proteolytic pathways
-
2+-activated, and ubiquitin-proteasome proteolytic pathways. J Clin Invest 1996;97:1610-1617. These investigations provide a quantitative index of the contribution of various components of the different proteolytic pathways to the overall rate of protein degradation after injection of live Escherichia coli.
-
(1996)
J Clin Invest
, vol.97
, pp. 1610-1617
-
-
Voisin, L.1
Brueille, D.2
Combaret, L.3
Pouyet, C.4
Taillandier, D.5
Arousseau, E.6
-
28
-
-
0029924205
-
Increased mRNA levels for components of the lysosomal, Ca2+-activated, and ATP-ubiquitin-dependent proteolytic pathways from head trauma patients
-
Mansoor O, Beufrere B, Boirie Y, Rulliere C, Taillandier D, Aurousseau E, et al. Increased mRNA levels for components of the lysosomal, Ca2+-activated, and ATP-ubiquitin-dependent proteolytic pathways from head trauma patients. Proc Natl Acad Sci USA 1996; 93:2714-2718.
-
(1996)
Proc Natl Acad Sci USA
, vol.93
, pp. 2714-2718
-
-
Mansoor, O.1
Beufrere, B.2
Boirie, Y.3
Rulliere, C.4
Taillandier, D.5
Aurousseau, E.6
-
29
-
-
0029164710
-
Role of oxidized amino acids in protein breakdown and stability
-
Stadtman E. Role of oxidized amino acids in protein breakdown and stability. Methods Enzymol 1995; 258:379-393.
-
(1995)
Methods Enzymol
, vol.258
, pp. 379-393
-
-
Stadtman, E.1
-
30
-
-
0023655258
-
Oxygen radicals stimulate intracellular proteolysis and lipid peroxidation by independent mechanisms in erythrocytes
-
Davies KJA, Goldberg AL. Oxygen radicals stimulate intracellular proteolysis and lipid peroxidation by independent mechanisms in erythrocytes. J Biol Chem 1987; 262:8220-8226.
-
(1987)
J Biol Chem
, vol.262
, pp. 8220-8226
-
-
Davies, K.J.A.1
Goldberg, A.L.2
-
31
-
-
0029853517
-
Sepsis increases oxidatively damaged proteins in skeletal muscle
-
Fagan JM, Ganguly M, Tiao G, Fischer JE, Hasselgren P-O. Sepsis increases oxidatively damaged proteins in skeletal muscle. Arch Surg 1996; 131:1326-1331.
-
(1996)
Arch Surg
, vol.131
, pp. 1326-1331
-
-
Fagan, J.M.1
Ganguly, M.2
Tiao, G.3
Fischer, J.E.4
Hasselgren, P.-O.5
-
32
-
-
0027460118
-
Glucocorticoids activate the ATP-ubiquitin-dependent proteolytic system in skeletal muscle during fasting
-
Wing S, Goldberg AL. Glucocorticoids activate the ATP-ubiquitin-dependent proteolytic system in skeletal muscle during fasting. Am J Physiol 1993; 264:E668-E676.
-
(1993)
Am J Physiol
, vol.264
-
-
Wing, S.1
Goldberg, A.L.2
-
33
-
-
0025973730
-
Cytokines and glucocorticoids in the regulation of the 'hepto-skeletal muscle axis' in sepsis
-
Fischer JE, Hasselgren PO. Cytokines and glucocorticoids in the regulation of the 'hepto-skeletal muscle axis' in sepsis. Arch Surg 1991; 161:266-271.
-
(1991)
Arch Surg
, vol.161
, pp. 266-271
-
-
Fischer, J.E.1
Hasselgren, P.O.2
-
34
-
-
0025761863
-
Effect of the glucocorticoid receptor RU 38486 on muscle protein breakdown in sepsis
-
Hall-Angeras M, Angeras U, Zamir O, Hasselgren PO, Fischer J. Effect of the glucocorticoid receptor RU 38486 on muscle protein breakdown in sepsis. Surgery 1991; 109:468-473.
-
(1991)
Surgery
, vol.109
, pp. 468-473
-
-
Hall-Angeras, M.1
Angeras, U.2
Zamir, O.3
Hasselgren, P.O.4
Fischer, J.5
-
35
-
-
0030051797
-
Energy-ubiqutin-dependent muscle proteolysis during sepsis is regulated by glucocorticoids
-
Tiao G, Fagan JM, Roegner V, Lieberman M, Wang JJ, Fischer JE, Hasselgren P-O. Energy-ubiqutin-dependent muscle proteolysis during sepsis is regulated by glucocorticoids. J Clin Invest 1996; 97:339-348.
-
(1996)
J Clin Invest
, vol.97
, pp. 339-348
-
-
Tiao, G.1
Fagan, J.M.2
Roegner, V.3
Lieberman, M.4
Wang, J.J.5
Fischer, J.E.6
Hasselgren, P.-O.7
-
36
-
-
0030909411
-
Activation of ubiquitin pathway in rat skeletal muscle by catabolic doses of glucocorticoids
-
Auclair D, Garrel DR, Zerouala AC, Ferland LH. Activation of ubiquitin pathway in rat skeletal muscle by catabolic doses of glucocorticoids. Am J Physiol 1997; 272:C1007-C1016. Administration of dexamethasome to animals activated the ubiqutin pathway of proteolysis in rat skeletal muscle, providing additional evidence for a role of glucocorticoids in initiating the changes in proteolysis in sepsis.
-
(1997)
Am J Physiol
, vol.272
-
-
Auclair, D.1
Garrel, D.R.2
Zerouala, A.C.3
Ferland, L.H.4
-
37
-
-
0028899142
-
Effects of dexamethasone on protein degradation and protease gene expression in rat L8 myotubes in culture
-
Hong DH, Forsberg NE. Effects of dexamethasone on protein degradation and protease gene expression in rat L8 myotubes in culture. Mol Cell Endocrinol 1995; 108:199-209.
-
(1995)
Mol Cell Endocrinol
, vol.108
, pp. 199-209
-
-
Hong, D.H.1
Forsberg, N.E.2
-
38
-
-
0026793224
-
Muscle protein breakdown during endotoxemia in rats after treament with interleukin-1 receptor antagonist
-
Zamir O, Hasselgren PO, O'Brien W, Thompson RC, Fischer JE. Muscle protein breakdown during endotoxemia in rats after treament with interleukin-1 receptor antagonist. Ann Surg 1992; 216:381-386.
-
(1992)
Ann Surg
, vol.216
, pp. 381-386
-
-
Zamir, O.1
Hasselgren, P.O.2
O'Brien, W.3
Thompson, R.C.4
Fischer, J.E.5
-
39
-
-
0027456605
-
In vivo administration of IL-1α induces muscle proteolysis in normal and adrenalectomized rats
-
Zamir O, Hasselgren PO, O'Brien W, Von Allmen D, Fischer JE. In vivo administration of IL-1α induces muscle proteolysis in normal and adrenalectomized rats. Metabolism 1993; 42:204-208.
-
(1993)
Metabolism
, vol.42
, pp. 204-208
-
-
Zamir, O.1
Hasselgren, P.O.2
O'Brien, W.3
Von Allmen, D.4
Fischer, J.E.5
-
40
-
-
0000863905
-
Tumor necrosis factor (TNF) and interleukin-1 (IL-1) induce muscle proteolysis through different mechanisms
-
Zamir O, Hasselgren PO, Higashiguchi T, Frederick JA, Fischer JE. Tumor necrosis factor (TNF) and interleukin-1 (IL-1) induce muscle proteolysis through different mechanisms. Mediat Inflam 1992; 1:247-250.
-
(1992)
Mediat Inflam
, vol.1
, pp. 247-250
-
-
Zamir, O.1
Hasselgren, P.O.2
Higashiguchi, T.3
Frederick, J.A.4
Fischer, J.E.5
-
41
-
-
0028201322
-
Interleukin-6 induces skeletal muscle protein breakdown in rats
-
Goodman MN. Interleukin-6 induces skeletal muscle protein breakdown in rats. Proc Soc Exp Biol Med 1994; 205:182-185.
-
(1994)
Proc Soc Exp Biol Med
, vol.205
, pp. 182-185
-
-
Goodman, M.N.1
-
42
-
-
0028789750
-
Interleukin-6 induces proteolysis by activating intracellula cathepsin B and L in C2C12 myotubes
-
Ebisui C, Tsujinaka T, Morimoto T, Kan K, Ijima S, Yano M, et al. Interleukin-6 induces proteolysis by activating intracellula cathepsin B and L in C2C12 myotubes. Clin Sci 1995; 89:431-439.
-
(1995)
Clin Sci
, vol.89
, pp. 431-439
-
-
Ebisui, C.1
Tsujinaka, T.2
Morimoto, T.3
Kan, K.4
Ijima, S.5
Yano, M.6
-
43
-
-
13344261948
-
Interleukin-6 antibody inhibits muscle atrophy and modulates proteolytic systems in interleukin-6 transgenic mice
-
Tsujinaka T, Fujita J, Ebisui C, Yano M, Kominami E, Suzuki K, et al. Interleukin-6 antibody inhibits muscle atrophy and modulates proteolytic systems in interleukin-6 transgenic mice. J Clin Invest 1996; 97:244-249.
-
(1996)
J Clin Invest
, vol.97
, pp. 244-249
-
-
Tsujinaka, T.1
Fujita, J.2
Ebisui, C.3
Yano, M.4
Kominami, E.5
Suzuki, K.6
-
45
-
-
0023752178
-
Insulin-like growth factor 1 production is inhibited in human sepsis
-
Dann MS, Lange LA, Jacobs LA. Insulin-like growth factor 1 production is inhibited in human sepsis. Arch Surg 1988; 123:1409-1414.
-
(1988)
Arch Surg
, vol.123
, pp. 1409-1414
-
-
Dann, M.S.1
Lange, L.A.2
Jacobs, L.A.3
-
46
-
-
0028103767
-
Insulin-like growth factor 1: Molecular biology and relevance to tissue-specific expression and action
-
Lund PK. Insulin-like growth factor 1: molecular biology and relevance to tissue-specific expression and action. Rec Prog Hormone Res 1994; 49:125-148.
-
(1994)
Rec Prog Hormone Res
, vol.49
, pp. 125-148
-
-
Lund, P.K.1
-
47
-
-
0028958031
-
Insulin-like growth factors and their binding proteins: Biological actions
-
Jones JI, Clemmons DR. Insulin-like growth factors and their binding proteins: biological actions. Endocr Rev 1995; 16:3-34.
-
(1995)
Endocr Rev
, vol.16
, pp. 3-34
-
-
Jones, J.I.1
Clemmons, D.R.2
-
48
-
-
0027357339
-
Insulin-like growth factor binding proteins
-
Rechter. MM. Insulin-like growth factor binding proteins. Vitam Horm 1993; 47:1-113.
-
(1993)
Vitam Horm
, vol.47
, pp. 1-113
-
-
Rechter, M.M.1
-
49
-
-
0028202862
-
Differential tissue regulation of insulin-like growth factor-I content and binding proteins after endotoxin
-
Fan J, Molina PE, Gelato MC, Lang CH. Differential tissue regulation of insulin-like growth factor-I content and binding proteins after endotoxin. Endocrinology 1994; 134:1685-1692.
-
(1994)
Endocrinology
, vol.134
, pp. 1685-1692
-
-
Fan, J.1
Molina, P.E.2
Gelato, M.C.3
Lang, C.H.4
-
50
-
-
0029984415
-
Interleukin-1 receptor antagonist attenuates sepsis-induced alterations in insulin-like growth factor system and protein synthesis
-
Lang CH, Fan J, Cooney R, Vary TC. Interleukin-1 receptor antagonist attenuates sepsis-induced alterations in insulin-like growth factor system and protein synthesis. Am J Physiol 1996; 270:E430-E437. These studies provide evidence for a role of IL-1 as an endogenous mediator of the sepsis-induced changes in the IGF-I system, and suggest that the accompanying changes in muscle protein synthesis are partially mediated by decreases in IGF-I.
-
(1996)
Am J Physiol
, vol.270
-
-
Lang, C.H.1
Fan, J.2
Cooney, R.3
Vary, T.C.4
-
51
-
-
0028945843
-
Alterations in the insulin-like growth factor system in trauma patients
-
Wojnar MM, Fan J, Frost RA, Gelato MC, Lang CH. Alterations in the insulin-like growth factor system in trauma patients. Am J Physiol 1995; 268:R970-R977.
-
(1995)
Am J Physiol
, vol.268
-
-
Wojnar, M.M.1
Fan, J.2
Frost, R.A.3
Gelato, M.C.4
Lang, C.H.5
-
52
-
-
0030785662
-
Acute alterations in growth hormone-insulin-like growth factor axis in humans injected with endotoxin
-
Lang CH, Pollard V, Fan J, Traber LD, Frost RA, Gelato MC, Prough DS. Acute alterations in growth hormone-insulin-like growth factor axis in humans injected with endotoxin. Am J Physiol 1997; 273:R371-R378.
-
(1997)
Am J Physiol
, vol.273
-
-
Lang, C.H.1
Pollard, V.2
Fan, J.3
Traber, L.D.4
Frost, R.A.5
Gelato, M.C.6
Prough, D.S.7
-
53
-
-
0005957168
-
Differential role of glucocorticoids in mediating endotoxin-induced changes in IGF-I and IGFBP-I
-
Li YH, Fan J, Lang CH. Differential role of glucocorticoids in mediating endotoxin-induced changes in IGF-I and IGFBP-I. Am J Physiol 1997; 272:R1990-R1997. These studies show that inhibition of glucocorticoids with a specific receptor antagonist failed to prevent lipopolysaccharide-induced decrease in IGF-I content in muscle.
-
(1997)
Am J Physiol
, vol.272
-
-
Li, Y.H.1
Fan, J.2
Lang, C.H.3
-
54
-
-
0028967330
-
The effects of insulin-like growth factor-I on protein metabolism and hepatic response to endotoxin in parenterally fed rats
-
Dickerson RN, Manzo CB, Charland SL, Settle RG, Stein TP, Kuhl DA, Rajter J-J. The effects of insulin-like growth factor-I on protein metabolism and hepatic response to endotoxin in parenterally fed rats. J Surg Res 1995; 58:260-266.
-
(1995)
J Surg Res
, vol.58
, pp. 260-266
-
-
Dickerson, R.N.1
Manzo, C.B.2
Charland, S.L.3
Settle, R.G.4
Stein, T.P.5
Kuhl, D.A.6
Rajter, J.-J.7
-
55
-
-
0028886248
-
Insulin-like growth factor-I accelerates protein synthesis in skeletal muscle during sepsis
-
Jurasinski CV, Vary TC. Insulin-like growth factor-I accelerates protein synthesis in skeletal muscle during sepsis. Am J Physiol 1995; 269:E977-E981.
-
(1995)
Am J Physiol
, vol.269
-
-
Jurasinski, C.V.1
Vary, T.C.2
-
56
-
-
0031261994
-
Cytokines block the effects of insulin-like growth factor-I (IGF-I) on glucose uptake and lactate production in skeletal muscle but do not influence IGF-I-induced changes in protein turnover
-
Fang C-H, Li BG, James JH, Fischer JE, Hasseigren P-O. Cytokines block the effects of insulin-like growth factor-I (IGF-I) on glucose uptake and lactate production in skeletal muscle but do not influence IGF-I-induced changes in protein turnover. Shock 1997; 8:362-367.
-
(1997)
Shock
, vol.8
, pp. 362-367
-
-
Fang, C.-H.1
Li, B.G.2
James, J.H.3
Fischer, J.E.4
Hasseigren, P.-O.5
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