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Volumn 41, Issue 2, 1998, Pages 237-240

Insulin selectively primes Th2 responses and induces regulatory tolerance to insulin in pre-diabetic mice

Author keywords

Autoimmunity; Diabetes; Immunotherapy; Insulin; NOD; Self antigen; Th1; Th2; Tolerance

Indexed keywords

HUMAN INSULIN;

EID: 0031888650     PISSN: 0012186X     EISSN: None     Source Type: Journal    
DOI: 10.1007/s001250050896     Document Type: Article
Times cited : (28)

References (10)
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  • 2
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    • Spontaneous loss of T-cell tolerance to glutamic acid decarboxylase in murine insulin-dependent diabetes
    • Kaufman DL, Clare-Salzler M, Tian J et al. (1993) Spontaneous loss of T-cell tolerance to glutamic acid decarboxylase in murine insulin-dependent diabetes. Nature 366: 69-72
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    • Kaufman, D.L.1    Clare-Salzler, M.2    Tian, J.3
  • 5
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    • Cellular immune response to diverse islet cell antigens in IDDM
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    • Schloot, N.C.1    Roep, B.O.2    Wegmann, D.3
  • 7
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    • Insulin antibody response to a short course of human insulin therapy in women with gestational diabetes
    • Balsells M, Garcia-Patterson A, Corcoy R et al. (1997) Insulin antibody response to a short course of human insulin therapy in women with gestational diabetes. Diabetes Care 20: 1172-1175
    • (1997) Diabetes Care , vol.20 , pp. 1172-1175
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  • 8
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    • Selective development of T helper (Th)2 cells induced by continuous administration of low dose soluble proteins to normal and beta(2)-microglobulin-deficient BALB/c mice
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  • 9
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    • Modulating autoimmune responses to GAD inhibits disease progression and prolongs islet graft survival in diabetes-prone mice
    • Tian J, Clare-Salzler M, Herschenfeld A et al. (1996) Modulating autoimmune responses to GAD inhibits disease progression and prolongs islet graft survival in diabetes-prone mice. Nat Med 2: 1348-1353
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.