Deoxygenation of sickle red blood cells stimulates KCl cotransport without affecting Na+/H+ exchange

American Journal of Physiology - Cell Physiology

Volumn 274, Issue 6 43-6, 1998, Pages

  Joiner, C H ^a,c   Jiang, M ^a   Fathallah, H ^b   Giraud, F ^b   Franco, R S ^a  

^a UNIVERSITY OF CINCINNATI COLLEGE OF MEDICINE   (United States)

^b CNRS   (France)

^c CINCINNATI CHILDREN'S HOSPITAL MEDICAL CENTER   (United States)

Author keywords

Erythrocyte; Potassium; Reticulocyte; Sodium; Volume regulation

Indexed keywords

CALCIMYCIN; CATION; HEMOGLOBIN S; MAGNESIUM ION; POTASSIUM ION; SODIUM ION;

ARTICLE; CELL VOLUME; DEPHOSPHORYLATION; ERYTHROCYTE; HUMAN; HUMAN CELL; ION EXCHANGE; ION TRANSPORT; MEMBRANE PERMEABILITY; POTASSIUM TRANSPORT; PRIORITY JOURNAL; PROTON SODIUM EXCHANGE; REOXYGENATION; RETICULOCYTE; SICKLE CELL; SODIUM TRANSPORT;

OXY;

EID: 0031774515     PISSN: 03636143     EISSN: None     Source Type: Journal    
DOI: 10.1152/ajpcell.1998.274.6.c1466     Document Type: Article

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