Cryptic brain cell injury caused by fetal nicotine exposure is associated with persistent elevations of c-fos protooncogene expression

Brain Research

Volumn 750, Issue 1-2, 1997, Pages 180-188

  Slotkin, T A ^a  

^a DUKE UNIVERSITY MEDICAL CENTER   (United States)

Author keywords

brain development; c fos protooncogene; nicotine

Indexed keywords

MESSENGER RNA; NICOTINE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN DEVELOPMENT; BRAIN INJURY; BRAIN REGION; CONTROLLED STUDY; FETUS; GENE EXPRESSION; NEWBORN; NONHUMAN; NORTHERN BLOTTING; ONCOGENE C FOS; PRENATAL EXPOSURE; PRIORITY JOURNAL; RAT;

AGING; ANALYSIS OF VARIANCE; ANIMALS; ANIMALS, NEWBORN; BRAIN; BRAIN STEM; DOSE-RESPONSE RELATIONSHIP, DRUG; EMBRYONIC AND FETAL DEVELOPMENT; FEMALE; GENE EXPRESSION REGULATION, DEVELOPMENTAL; GENES, FOS; GESTATIONAL AGE; NICOTINE; ORGAN SIZE; PREGNANCY; PRENATAL EXPOSURE DELAYED EFFECTS; PROSENCEPHALON; PROTO-ONCOGENE PROTEINS C-FOS; RATS; RATS, SPRAGUE-DAWLEY; RNA, MESSENGER; TRANSCRIPTION, GENETIC;

EID: 0031043422     PISSN: 00068993     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0006-8993(96)01345-5     Document Type: Article

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