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Volumn 49, Issue 3, 1997, Pages 890-892

Homozygotes and heterozygotes for ciliary neurotrophic factor null alleles do not show earlier onset of Huntington's disease

Author keywords

[No Author keywords available]

Indexed keywords

4 AMINOBUTYRIC ACID; 4 AMINOBUTYRIC ACID RECEPTOR; CILIARY NEUROTROPHIC FACTOR; EXCITOTOXIN; QUINOLINIC ACID;

EID: 0030826134     PISSN: 00283878     EISSN: None     Source Type: Journal    
DOI: 10.1212/WNL.49.3.890     Document Type: Article
Times cited : (4)

References (10)
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  • 2
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  • 3
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  • 4
    • 0028100732 scopus 로고
    • Molecular and clinical correlations in spinocerebellar ataxia type 1: Evidence for familial effects on the age at onset
    • Ranum LPW, Chung M-Y, Banfi S, et al. Molecular and clinical correlations in spinocerebellar ataxia type 1: evidence for familial effects on the age at onset. Am J Hum Genet 1994;55: 244-252.
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    • A null mutation in the human CNTF gene is not causally related to neurological diseases
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  • 7
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    • Implants of encapsulated human CNTF-producing fibroblasts prevent behavioural deficits and striatal degeneration in a rodent model of Huntington's disease
    • Emerich DF, Lindner MD, Winn SR, Chen EY, Frydel BR, Kordower JH. Implants of encapsulated human CNTF-producing fibroblasts prevent behavioural deficits and striatal degeneration in a rodent model of Huntington's disease. J Neurosci 1996;16:5168-5181.
    • (1996) J Neurosci , vol.16 , pp. 5168-5181
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.