-
1
-
-
0001862422
-
Afterdepolarizations and triggered activity: Distinction from automaticity as an arrhythmogenic mechanism
-
Fozzard HA, Haber E, Jennings RB, Katz AM, Morgan HE, eds. 2nd ed. New York: Raven Press
-
Wit AL, Rosen MR. Afterdepolarizations and triggered activity: distinction from automaticity as an arrhythmogenic mechanism. In: Fozzard HA, Haber E, Jennings RB, Katz AM, Morgan HE, eds. Heart and cardiovascular system: Scientific foundations, vol. 2, 2nd ed. New York: Raven Press, 1991:2113-63.
-
(1991)
Heart and Cardiovascular System: Scientific Foundations
, vol.2
, pp. 2113-2163
-
-
Wit, A.L.1
Rosen, M.R.2
-
2
-
-
0028082351
-
Clinical relevance of cardiac arrhythmias generated by afterdepolarizations. Role of M cells in the generation of U waves, triggered activity and torsade de points
-
Antzelevitch C, Sicouri S. Clinical relevance of cardiac arrhythmias generated by afterdepolarizations. Role of M cells in the generation of U waves, triggered activity and torsade de points. J Am Coll Cardiol 1994;23: 259-77.
-
(1994)
J Am Coll Cardiol
, vol.23
, pp. 259-277
-
-
Antzelevitch, C.1
Sicouri, S.2
-
3
-
-
0020501838
-
Bradycardia-dependent triggered activity: Relevance to drug-induced multiform ventricular tachycardia
-
Brachmann J, Scherlag BJ, Rosenshtraukh LV, Lazzara R. Bradycardia-dependent triggered activity: relevance to drug-induced multiform ventricular tachycardia. Circulation 1983;68:846-56.
-
(1983)
Circulation
, vol.68
, pp. 846-856
-
-
Brachmann, J.1
Scherlag, B.J.2
Rosenshtraukh, L.V.3
Lazzara, R.4
-
4
-
-
0023784610
-
QTU prolongation and polymorphic ventricular tachyarrhythmias due to bradycardia-dependent early afterdepolarizations. Afterdepolarizations and ventricular arrhythmias
-
el-Sherif N, Zeiler RH, Craelius W, Gough WB, Henkin R. QTU prolongation and polymorphic ventricular tachyarrhythmias due to bradycardia-dependent early afterdepolarizations. Afterdepolarizations and ventricular arrhythmias. Circ Res 1988;63:286-305.
-
(1988)
Circ Res
, vol.63
, pp. 286-305
-
-
El-Sherif, N.1
Zeiler, R.H.2
Craelius, W.3
Gough, W.B.4
Henkin, R.5
-
5
-
-
0030022457
-
Kr. Implications for torsade de pointes and reverse use-dependence
-
Kr. Implications for torsade de pointes and reverse use-dependence. Circulation 1996;93:407-11.
-
(1996)
Circulation
, vol.93
, pp. 407-411
-
-
Yang, T.1
Roden, D.M.2
-
6
-
-
0028881535
-
Ks contributes to the longer action potential of the M cell
-
Ks contributes to the longer action potential of the M cell. Circ Res 1995;76:351-65.
-
(1995)
Circ Res
, vol.76
, pp. 351-365
-
-
Liu, D.W.1
Antzelevitch, C.2
-
7
-
-
0029847602
-
Multiple mechanisms in the long-QT syndrome. Current knowledge, gaps, and future directions
-
Roden DM, Lazzara R, Rosen M, Schwartz PJ, Towbin J, Vincent GM. Multiple mechanisms in the long-QT syndrome. Current knowledge, gaps, and future directions. Circulation 1996;94:1996-2012.
-
(1996)
Circulation
, vol.94
, pp. 1996-2012
-
-
Roden, D.M.1
Lazzara, R.2
Rosen, M.3
Schwartz, P.J.4
Towbin, J.5
Vincent, G.M.6
-
8
-
-
16944362512
-
Four novel KVLQT1 and four novel HERG mutations in familial long-QT syndrome
-
Tanaka T, Nagai R, Tomoike H, et al. Four novel KVLQT1 and four novel HERG mutations in familial long-QT syndrome. Circulation 1997; 95:565-7.
-
(1997)
Circulation
, vol.95
, pp. 565-567
-
-
Tanaka, T.1
Nagai, R.2
Tomoike, H.3
-
11
-
-
0030918946
-
Molecular basis of the long-QT syndrome associated with deafness
-
Splawski I, Timothy KW, Vincent GM, et al. Molecular basis of the long-QT syndrome associated with deafness. N Engl J Med 1996;336: 1562-7.
-
(1996)
N Engl J Med
, vol.336
, pp. 1562-1567
-
-
Splawski, I.1
Timothy, K.W.2
Vincent, G.M.3
-
12
-
-
0028861892
-
ECG T-wave patterns in genetically distinct forms of the hereditary long QT syndrome
-
Moss AJ, Zareba W, Benhorin J, et al. ECG T-wave patterns in genetically distinct forms of the hereditary long QT syndrome. Circulation 1995;92:2929-34.
-
(1995)
Circulation
, vol.92
, pp. 2929-2934
-
-
Moss, A.J.1
Zareba, W.2
Benhorin, J.3
-
13
-
-
0028874658
-
Long QT syndrome patients with mutations of the SCN5A and HERG genes have differential responses to Na+ channel blockade and to increases in heart rate. Implications for gene-specific therapy
-
Schwartz PJ, Priori SG, Locati EH, et al. Long QT syndrome patients with mutations of the SCN5A and HERG genes have differential responses to Na+ channel blockade and to increases in heart rate. Implications for gene-specific therapy. Circulation 1995;92:3381-6.
-
(1995)
Circulation
, vol.92
, pp. 3381-3386
-
-
Schwartz, P.J.1
Priori, S.G.2
Locati, E.H.3
-
14
-
-
0029831629
-
Genetically defined therapy of inherited long-QT syndrome. Correction of abnormal repolarization by potassium
-
Comptom SJ, Lux RL, Ramsey MR, et al. Genetically defined therapy of inherited long-QT syndrome. Correction of abnormal repolarization by potassium. Circulation 1996;94:1018-22.
-
(1996)
Circulation
, vol.94
, pp. 1018-1022
-
-
Comptom, S.J.1
Lux, R.L.2
Ramsey, M.R.3
-
15
-
-
0028912946
-
Mechanisms of adenosine-mediated actions on cellular and clinical cardiac electrophysiology
-
Shen WK, Kurachi Y. Mechanisms of adenosine-mediated actions on cellular and clinical cardiac electrophysiology. Mayo Clin Proc 1995;70: 274-91.
-
(1995)
Mayo Clin Proc
, vol.70
, pp. 274-291
-
-
Shen, W.K.1
Kurachi, Y.2
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