Radiation-induced apoptosis in developing rats and kainic acid-induced excitotoxicity in adult rats are associated with distinctive morphological and biochemical c-Jun/AP-1 (N) expression

Neuroscience

Volumn 80, Issue 2, 1997, Pages 449-458

  Ferrer, I ^a   Planas, A M ^b   Pozas, E ^a  

^a UNIVERSITY OF BARCELONA   (Spain)

^b INST D INVESTIGACIONS B   (Spain)

Author keywords

Apoptosis; c Jun; Cell death; Ionizing radiation; JNK 1; Kainic acid

Indexed keywords

DNA FRAGMENT; KAINIC ACID; PHOSPHOTRANSFERASE; TRANSCRIPTION FACTOR AP 1;

ADOLESCENT; AMYGDALOID NUCLEUS; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; BRAIN DEVELOPMENT; CHROMATIN CONDENSATION; CONTROLLED STUDY; CYTOTOXICITY; ENTORHINAL CORTEX; ETIOLOGY; FEMALE; HIPPOCAMPUS; IMMUNOBLOTTING; IMMUNOHISTOCHEMISTRY; IONIZING RADIATION; MALE; NONHUMAN; ONCOGENE C JUN; PRIORITY JOURNAL; PROTEIN PROCESSING; PYRIFORM CORTEX; RAT; THALAMUS;

ANIMALS; APOPTOSIS; BLOTTING, WESTERN; BRAIN; DNA FRAGMENTATION; ELECTROPHORESIS, POLYACRYLAMIDE GEL; EXCITATORY AMINO ACID AGONISTS; FEMALE; GENE EXPRESSION REGULATION; IMMUNOHISTOCHEMISTRY; KAINIC ACID; MALE; PROTO-ONCOGENE PROTEINS C-JUN; RATS; RATS, SPRAGUE-DAWLEY; TRANSCRIPTION FACTOR AP-1;

EID: 0030810487     PISSN: 03064522     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0306-4522(97)00126-7     Document Type: Article

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