p57(KIP2) targeted disruption and Beckwith-Wiedemann syndrome: Is the inhibitor just a contributor?

BioEssays

Volumn 19, Issue 10, 1997, Pages 839-842

  Swanger, W Jherek ^a   Roberts, James M ^a  

^a Fred Hutchinson Cancer Research Center   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CDKN1C PROTEIN, HUMAN; CDKN1C PROTEIN, MOUSE; CYCLIN DEPENDENT KINASE INHIBITOR 1C; ENZYME INHIBITOR; NUCLEAR PROTEIN; SOMATOMEDIN B;

ANIMAL; BECKWITH WIEDEMANN SYNDROME; BIOSYNTHESIS; CELL CYCLE; GENETICS; HUMAN; MOUSE; MOUSE MUTANT; MULTIPLE MALFORMATION SYNDROME; REVIEW; TRANSGENIC MOUSE;

ABNORMALITIES, MULTIPLE; ANIMALS; BECKWITH-WIEDEMANN SYNDROME; CELL CYCLE; CYCLIN-DEPENDENT KINASE INHIBITOR P57; ENZYME INHIBITORS; HUMANS; INSULIN-LIKE GROWTH FACTOR II; MICE; MICE, KNOCKOUT; MICE, TRANSGENIC; NUCLEAR PROTEINS;

EID: 0030609139     PISSN: 02659247     EISSN: None     Source Type: Journal    
DOI: 10.1002/bies.950191002     Document Type: Article

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