Deletion and mutation analyses of the P16/MTS-1 tumor suppressor gene in human ductal pancreatic cancer reveals a higher frequency of abnormalities in tumor-derived cell lines than in primary ductal adenocarcinomas

Cancer Research

Volumn 56, Issue 5, 1996, Pages 1137-1141

  Huang, Lingyi ^a   Goodrow, Tamra L ^b   Zhang, Shi Yu ^b   Klein Szanto, Andres J P ^b   Chang, Hong ^c   Ruggeri, Bruce A ^a  

^a DREXEL UNIVERSITY COLLEGE OF MEDICINE   (United States)

^b FOX CHASE CANCER CENTER   (United States)

^c NONE

Author keywords

[No Author keywords available]

Indexed keywords

ARTICLE; CELL PROLIFERATION; DNA SEQUENCE; EXON; GENE DELETION; GENE FREQUENCY; GENE MUTATION; GENETIC ANALYSIS; HUMAN; HUMAN CELL; MUTATION RATE; PANCREAS CARCINOMA; PANCREAS DUCT; PRIORITY JOURNAL; SINGLE STRAND CONFORMATION POLYMORPHISM; TUMOR SUPPRESSOR GENE;

ADENOCARCINOMA; CARRIER PROTEINS; CYCLIN-DEPENDENT KINASE INHIBITOR P16; DNA MUTATIONAL ANALYSIS; GENE DELETION; GENES, TUMOR SUPPRESSOR; HUMANS; PANCREATIC NEOPLASMS; TUMOR CELLS, CULTURED;

EID: 0030043385     PISSN: 00085472     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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