Lack of mutation in the cyclin-dependent kinase inhibitor, p19(INK4d), in tumor-derived cell lines and primary tumors

Oncogene

Volumn 13, Issue 9, 1996, Pages 2033-2038

  Zariwala, Maimoona ^a   Xiong, Yue ^a  

^a UNIVERSITY OF NORTH CAROLINA SCHOOL OF MEDICINE   (United States)

Author keywords

CDK inhibitors; p15(INK4b); p16(INK4a); p18(INK4c); p19(INK4d)

Indexed keywords

CYCLIN DEPENDENT KINASE; PROTEIN KINASE INHIBITOR;

ACUTE GRANULOCYTIC LEUKEMIA; ARTICLE; CARCINOGENESIS; CODON; ENZYME INHIBITION; EXON; GENETIC POLYMORPHISM; OVARY TUMOR; PRIORITY JOURNAL; TUMOR CELL LINE; TUMOR SUPPRESSOR GENE;

CARRIER PROTEINS; CELL CYCLE PROTEINS; CELL LINE; CELL LINE, TRANSFORMED; CYCLIN-DEPENDENT KINASE INHIBITOR P15; CYCLIN-DEPENDENT KINASE INHIBITOR P16; CYCLIN-DEPENDENT KINASE INHIBITOR P18; CYCLIN-DEPENDENT KINASE INHIBITOR P19; ENZYME INHIBITORS; FEMALE; FIBROBLASTS; GENE DELETION; HOMOZYGOTE; HUMANS; LEUKEMIA, MYELOCYTIC, ACUTE; MUTATION; NEOPLASMS; OVARIAN NEOPLASMS; POLYMERASE CHAIN REACTION; POLYMORPHISM, GENETIC; TUMOR CELLS, CULTURED; TUMOR SUPPRESSOR PROTEINS;

EID: 0029957422     PISSN: 09509232     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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