Genetic susceptibility to chemical-induced cancers: Methodologic problems

International Journal of Occupational and Environmental Health

Volumn 2, Issue 3 SUPPL., 1996, Pages

  Vineis, Paolo ^a   D'Errico, Angelo ^a,b  

^a UNIVERSITY OF TURIN   (Italy)

^b NEW YORK UNIVERSITY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

EID: 9844237210     PISSN: 10773525     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (10)

1. Ayesh R, Idle JR, Ritchie JC, Crothers MJ, Hetzel MR. Metabolic oxidation phenotypes as markers of lung cancer susceptibility. Nature. 1984;312:169-70.
2. D'Errico A, Vineis P. Methodological problems in human studies of metabolic polymorphisms and cancer. [Manuscript in preparation.]
3. Vineis P, Bartsch H, Caporaso N, et al. Genetically based N-acetyltransferase metabolic polymorphism and low-level environmental exposure to carcinogens. Nature. 1994;369:154-6.
4. Taioli E, Kinney P, Zhitovic A, et al. Application of reliability models to studies of biomarker validation. Environ Health Perspect. 1994;102:306-9.
5. Pierce N. What does the odds ratio estimate in a case-control study? Int J Epidemiol. 1993;22:1189-92.
6. Axelson O, Fredriksson M, Ekberg K. Use of the prevalence ratio vs. the prevalence odds ratio as a measure of risk in cross-sectional studies. (letter). Occup Environ Med. 1994;51:574.
7. Cartwright R, Ahmad RA, Barham-Hall D, et al. Role of N-acetyltransferase phenotypes in bladder carcinogenesis: a pharmacogenetic epidemiological approach to bladder cancer. Lancet. 1982;ii:842-5.
8. Hayes RB, Bi W, Rothman N, et al. N-Acetylation phenotype and genotype and risk of bladder cancer in benzidine-exposed workers. Carcinogenesis. 1993;14.675-8.
9. Caporaso N, Landi MT, Vineis P. Relevance of metabolic polymorphisms to human carcinogenesis: evaluation of epidemiologic evidence. Pharmacogenetics. 1991;1:4-19.
10. Vineis P, McMichael A. Interplay between heterocyclic amines in cooked meat and metabolic phenotype in the etiology of colon cancer. [Submitted for publication.]