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Volumn 18, Issue 13, 2004, Pages 1606-1608
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A new strategy to block tumor growth by inhibiting endocannabinoid inactivation
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Author keywords
[No Author keywords available]
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Indexed keywords
AA 5 HT;
ANANDAMIDE;
ANTINEOPLASTIC AGENT;
ARVANIL;
CANNABINOID;
CANNABINOID RECEPTOR;
CANNABINOID RECEPTOR AGONIST;
FATTY ACID AMIDASE;
N (4 HYDROXY 2 METHYLPHENYL)ARACHIDONAMIDE;
UNCLASSIFIED DRUG;
VANILLOID RECEPTOR;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANIMAL TISSUE;
ARTICLE;
CANCER CONTROL;
CANCER INHIBITION;
CELL PROLIFERATION;
DRUG DEGRADATION;
DRUG EFFICACY;
DRUG HYDROLYSIS;
DRUG RECEPTOR BINDING;
DRUG TUMOR LEVEL;
MOUSE;
NONHUMAN;
PRIORITY JOURNAL;
RAT;
THYROID TUMOR;
TUMOR GROWTH;
TUMOR VOLUME;
ANIMALS;
ARACHIDONIC ACIDS;
BIODEGRADATION, ENVIRONMENTAL;
CELL PROLIFERATION;
ENDOCANNABINOIDS;
MICE;
MICE, NUDE;
PIPERIDINES;
PYRAZOLES;
RATS;
RECEPTOR, CANNABINOID, CB1;
SEROTONIN;
THYROID NEOPLASMS;
XENOGRAFT MODEL ANTITUMOR ASSAYS;
ANIMALIA;
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EID: 9444280832
PISSN: 08926638
EISSN: None
Source Type: Journal
DOI: 10.1096/fj.04-1754fje Document Type: Article |
Times cited : (144)
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References (0)
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