Reversal of the transformed phenotype and inhibition of peptidylglycine α-monooxygenase in Ras-transformed cells by 4-phenyl-3-butenoic acid

Molecular Carcinogenesis

Volumn 41, Issue 4, 2004, Pages 231-246

  Sunman, Jeffrey A ^a   Foster, Michael S ^b   Folse, Stacey L ^a   May, Sheldon W ^b   Matesic, Diane F ^a  

^a MERCER UNIVERSITY   (United States)

^b GEORGIA INSTITUTE OF TECHNOLOGY   (United States)

Author keywords

Amidation; Connexin; Oncogene; Phosphorylation

Indexed keywords

4 PHENYL 3 BUTENOIC ACID; ALPHA AMIDATING ENZYME; CONNEXIN 43; MITOGEN ACTIVATED PROTEIN KINASE; OXYGENASE INHIBITOR; PROTEIN P21; PROTEIN P42; PROTEIN P44; RAS PROTEIN; UNCLASSIFIED DRUG;

ANIMAL CELL; ARTICLE; CELL COMMUNICATION; CELL GROWTH; CELL PROLIFERATION; CELL STRUCTURE; CELL TRANSFORMATION; CELLULAR DISTRIBUTION; CONCENTRATION RESPONSE; CONTROLLED STUDY; CYTOTOXICITY; ENZYME ACTIVITY; ENZYME INHIBITION; ENZYME PHOSPHORYLATION; GAP JUNCTION; GROWTH INHIBITION; LIVER CELL; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; RAT; UPREGULATION; WESTERN BLOTTING;

CELL LINE, TRANSFORMED; CELL PROLIFERATION; CELL TRANSFORMATION, NEOPLASTIC; CONNEXIN 43; FATTY ACIDS, MONOUNSATURATED; GAP JUNCTIONS; GENE EXPRESSION REGULATION; HUMANS; MITOGEN-ACTIVATED PROTEIN KINASE 1; MITOGEN-ACTIVATED PROTEIN KINASE 3; MIXED FUNCTION OXYGENASES; MULTIENZYME COMPLEXES; ONCOGENE PROTEIN P21(RAS); PHENOTYPE; PHOSPHORYLATION; PROTEIN TRANSPORT; SIGNAL TRANSDUCTION; TIME FACTORS;

EID: 9244237032     PISSN: 08991987     EISSN: None     Source Type: Journal    
DOI: 10.1002/mc.20060     Document Type: Article

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