High levels of circulating β-amyloid peptide do not cause cerebral β- amyloidosis in transgenic mice

American Journal of Pathology

Volumn 149, Issue 1, 1996, Pages 219-227

  Fukuchi, Ken Ichiro ^a,b   Ho, Libin ^c,d   Younkin, Steven G ^c,d   Kunkel, Dennis D ^a   Ogburn, Charles E ^a   LeBoeuf, Renee C ^a   Furlong, Clement E ^a   Deeb, Samir S ^a   Nochlin, David ^a   Wegiel, Jerzy ^e   Wisniewski, Henry M ^e   Martin, George M ^a  

^a UNIVERSITY OF WASHINGTON   (United States)

^b UNIVERSITY OF ALABAMA SCHOOL OF MEDICINE   (United States)

^c CASE WESTERN RESERVE UNIVERSITY   (United States)

^d MAYO CLINIC   (United States)

^e NEW YORK STATE INSTITUTE FOR BASIC RESEARCH IN DEVELOPMENTAL DISABILITIES   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

AMYLOID BETA PROTEIN; AMYLOID PRECURSOR PROTEIN;

AMYLOIDOSIS; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN DISEASE; CARBOXY TERMINAL SEQUENCE; CONTROLLED STUDY; ENTEROPATHY; MOUSE; NONHUMAN; PRIORITY JOURNAL; TRANSGENIC MOUSE;

AMYLOID BETA-PROTEIN; AMYLOIDOSIS; ANIMALS; BLOTTING, SOUTHERN; BLOTTING, WESTERN; CEREBRAL CORTEX; DISEASE MODELS, ANIMAL; ELECTROPHORESIS, POLYACRYLAMIDE GEL; FEMALE; IMMUNOHISTOCHEMISTRY; INTESTINE, SMALL; MALE; MICE; MICE, TRANSGENIC; MYOCARDIUM; NUCLEIC ACID HYBRIDIZATION; PEPTIDE FRAGMENTS; RNA, MESSENGER;

EID: 8944228919     PISSN: 00029440     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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