Soluble fms-like tyrosine kinase 1 [7]

New England Journal of Medicine

Volumn 351, Issue 21, 2004, Pages 2241-2242

  Stepan, Holger ^a   Geide, Anne ^a   Faber, Renaldo ^a  

^a UNIVERSITY OF LEIPZIG   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

VASCULOTROPIN; VASCULOTROPIN RECEPTOR 1;

COHORT ANALYSIS; DOPPLER FLOWMETRY; HUMAN; HYPERTENSION; INTRAUTERINE GROWTH RETARDATION; LETTER; PREECLAMPSIA; PREGNANCY; PREGNANCY COMPLICATION; PRIORITY JOURNAL; PROTEIN BLOOD LEVEL; RISK FACTOR; SOLUBILITY;

FEMALE; FETAL GROWTH RETARDATION; HUMANS; PILOT PROJECTS; PRE-ECLAMPSIA; PREGNANCY; UTERUS; VASCULAR ENDOTHELIAL GROWTH FACTOR RECEPTOR-1;

EID: 8644234246     PISSN: 00284793     EISSN: None     Source Type: Journal    
DOI: 10.1056/NEJM200411183512123     Document Type: Letter

References (3)

1. Maynard SE, Min Y, Merchan J, et al. Excess placental soluble fms-like tyrosine kinase (sFlt1) may contribute to endothelial dysfunction, hypertension, and proteinuria in preeclampsia. J Clin Invest 2003;111:649-58.
2. Levine RJ, Maynard SE, Qian C, et al. Circulating angiogenic factors and the risk of preeclampsia. N Engl J Med 2004;350:672-83.
3. Chien PF, Arnott N, Gordon A, Owen P, Khan KS. How useful is uterine artery Doppler flow velocimetry in the prediction of preeclampsia, intrauterine growth retardation and perinatal death? An overview. BJOG 2000;107:196-208