Chronic Lithium Treatment Increases Telomere Length in Parietal Cortex and Hippocampus of Triple-Transgenic Alzheimer's Disease Mice

Journal of Alzheimer's disease : JAD

Volumn 63, Issue 1, 2018, Pages 93-101

  Cardillo, Giancarlo de Mattos ^a   De Paula, Vanessa de Jesus Rodrigues ^a   Ikenaga, Eliza Hiromi ^a   Costa, Luciana Rodrigues ^a   Catanozi, Sergio ^a   Schaeffer, Evelin Lisete ^a   Gattaz, Wagner Farid ^a   Kerr, Daniel Shikanai ^a,b   Forlenza, Orestes Vicente ^a  

^a UNIVERSITY OF SÃO PAULO   (Brazil)

^b Docente Do Instituto Federal Catarinense   (Brazil)

Author keywords

Alzheimer s disease; hippocampus; lithium; olfactory epithelium; parietal cortex; telomere; transgenic mice

Indexed keywords

AMYLOID PRECURSOR PROTEIN; LITHIUM DERIVATIVE; NEUROLEPTIC AGENT; PRESENILIN 1; PSEN1 PROTEIN, HUMAN; TAU PROTEIN;

ALZHEIMER DISEASE; ANIMAL; BLOOD; C57BL MOUSE; DISEASE MODEL; DRUG EFFECT; GENETICS; HIPPOCAMPUS; HUMAN; MALE; METABOLISM; MOUSE; MUTATION; PARIETAL LOBE; PATHOLOGY; TELOMERE HOMEOSTASIS; TRANSGENIC MOUSE;

ALZHEIMER DISEASE; AMYLOID BETA-PROTEIN PRECURSOR; ANIMALS; ANTIPSYCHOTIC AGENTS; DISEASE MODELS, ANIMAL; HIPPOCAMPUS; HUMANS; LITHIUM COMPOUNDS; MALE; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; MUTATION; PARIETAL LOBE; PRESENILIN-1; TAU PROTEINS; TELOMERE HOMEOSTASIS;

EID: 85060910289     PISSN: None     EISSN: 18758908     Source Type: Journal    
DOI: 10.3233/JAD-170838     Document Type: Article

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