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Volumn 21, Issue 7, 2002, Pages 990-999
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A conserved intronic response element mediates direct p53-dependent transcriptional activation of both the human and murine bax genes
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Author keywords
bax; DNA binding; p53; Transcription
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Indexed keywords
MESSENGER RNA;
MUTANT PROTEIN;
PROTEIN BAX;
PROTEIN P53;
ANIMAL CELL;
APOPTOSIS;
ARTICLE;
BINDING SITE;
CONTROLLED STUDY;
DNA RESPONSIVE ELEMENT;
ELECTROPHORETIC MOBILITY;
GENE DELETION;
GENE EXPRESSION REGULATION;
GENETIC CONSERVATION;
HUMAN;
HUMAN CELL;
INTRON;
MOUSE;
NONHUMAN;
NUCLEOTIDE SEQUENCE;
PRIORITY JOURNAL;
PROMOTER REGION;
PROTEIN DNA BINDING;
REPORTER GENE;
TRANSCRIPTION INITIATION;
TUMOR SUPPRESSOR GENE;
ANIMALS;
BASE SEQUENCE;
BCL-2-ASSOCIATED X PROTEIN;
CLONING, MOLECULAR;
CONSERVED SEQUENCE;
ELECTROPHORETIC MOBILITY SHIFT ASSAY;
GENES;
GENES, REPORTER;
HUMANS;
INTRONS;
MICE;
PROTO-ONCOGENE PROTEINS;
PROTO-ONCOGENE PROTEINS C-BCL-2;
RESPONSE ELEMENTS;
RNA, MESSENGER;
SEQUENCE ALIGNMENT;
TRANS-ACTIVATION (GENETICS);
TUMOR CELLS, CULTURED;
TUMOR SUPPRESSOR PROTEIN P53;
ANIMALIA;
MURINAE;
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EID: 85047697335
PISSN: 09509232
EISSN: None
Source Type: Journal
DOI: 10.1038/sj.onc.1205069 Document Type: Article |
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Times cited : (114)
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References (29)
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