Crowd Control: E7 Conservation Is the Key to Cancer

Cell

Volumn 170, Issue 6, 2017, Pages 1057-1059

  White, Elizabeth A ^a   Munger, Karl ^b  

^a UNIVERSITY OF PENNSYLVANIA   (United States)

^b TUFTS UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

PROTEIN E7; ONCOPROTEIN; REPRESSOR PROTEIN;

CANCER GROWTH; CANCER PATIENT; CANCER RISK; CARCINOGENESIS; DNA SYNTHESIS; E7 GENE; ENZYME ACTIVITY; GENE FUNCTION; GENE MUTATION; GENETIC CONSERVATION; GENETIC VARIABILITY; HIGH RISK PATIENT; HIGH THROUGHPUT SEQUENCING; HUMAN; HUMAN PAPILLOMAVIRUS DNA TEST; HUMAN PAPILLOMAVIRUS TYPE 16; NONHUMAN; PRIORITY JOURNAL; PROTEIN FUNCTION; SHORT SURVEY; SIGNAL TRANSDUCTION; UTERINE CERVIX CANCER; VIRUS ISOLATION; VIRUS REPLICATION; NEOPLASM;

HUMAN PAPILLOMAVIRUS 16; HUMANS; NEOPLASMS; ONCOGENE PROTEINS, VIRAL; PAPILLOMAVIRUS E7 PROTEINS; REPRESSOR PROTEINS;

EID: 85028956791     PISSN: 00928674     EISSN: 10974172     Source Type: Journal    
DOI: 10.1016/j.cell.2017.08.033     Document Type: Short Survey

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