Mechanism of C5a-induced immunologic derangement in sepsis

Cellular and Molecular Immunology

Volumn 14, Issue 9, 2017, Pages 792-793

  Xu, Ruonan ^a   Lin, Fang ^b   Bao, Chunmei ^c   Wang, Fu Sheng ^a  

^a BEIJING 302 HOSPITAL   (China)

^b Institute of Intensive Care Unit   (China)

^c Institute of Clinical Examination Centre   (China)

Author keywords

[No Author keywords available]

Indexed keywords

BIOLOGICAL MARKER; COMPLEMENT COMPONENT C5A; COMPLEMENT COMPONENT C5A RECEPTOR;

APOPTOSIS; BACTEREMIA; CELL PH; COMPLEMENT ACTIVATION; CORONAVIRIDAE; CYTOKINE RELEASE; DISEASE SEVERITY; HUMAN; IMMUNE DYSREGULATION; IMMUNE RESPONSE; IMMUNOPARALYSIS; IMMUNOPATHOGENESIS; INFLUENZA A VIRUS (H5N1); INFLUENZA A VIRUS (H7N9); LETTER; LEUKOCYTE ACTIVATION; LYMPHOCYTE; MICROVASCULATURE; MOLECULAR PATHOLOGY; NEUTROPHIL; NEUTROPHIL CHEMOTAXIS; NONHUMAN; PHAGOCYTOSIS; PROGNOSIS; PROTEIN EXPRESSION; RECEPTOR BINDING; RECEPTOR BLOCKING; SEPSIS; THYMOCYTE; ANIMAL; IMMUNOLOGY; METABOLISM; PATHOLOGY;

ANIMALS; BACTEREMIA; COMPLEMENT ACTIVATION; COMPLEMENT C5A; HUMANS; NEUTROPHILS; RECEPTOR, ANAPHYLATOXIN C5A; SEPSIS;

EID: 85028711097     PISSN: 16727681     EISSN: None     Source Type: Journal    
DOI: 10.1038/cmi.2017.68     Document Type: Letter

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