DNA immunization against amyloid beta 42 has high potential as safe therapy for Alzheimer's disease as it diminishes antigen-specific Th1 and Th17 cell proliferation.

Cellular and molecular neurobiology

Volumn 31, Issue 6, 2011, Pages 867-874

  Lambracht Washington, Doris ^a   Qu, Bao Xi ^b   Fu, Min ^b   Anderson Jr , Larry D ^b   Stüve, Olaf ^b   Eagar, Todd N ^b   Rosenberg, Roger N ^b  

^a UNIVERSITY OF TEXAS SOUTHWESTERN MEDICAL CENTER   (United States)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

5 (6) CARBOXYFLUORESCEIN DIACETATE SUCCINIMIDYL ESTER; 5-(6)-CARBOXYFLUORESCEIN DIACETATE SUCCINIMIDYL ESTER; AMYLOID BETA PROTEIN; CYTOKINE; DNA; EPITOPE; FLUORESCEIN DERIVATIVE; SUCCINIMIDE DERIVATIVE;

ALZHEIMER DISEASE; ANIMAL; ARTICLE; CELL PROLIFERATION; CYTOLOGY; ENZYME LINKED IMMUNOSPOT ASSAY; HUMORAL IMMUNITY; IMMUNIZATION; IMMUNOLOGY; MOUSE; SECRETION; TH1 CELL; TH17 CELL;

ALZHEIMER DISEASE; AMYLOID BETA-PEPTIDES; ANIMALS; CELL PROLIFERATION; CYTOKINES; DNA; ENZYME-LINKED IMMUNOSPOT ASSAY; EPITOPES; FLUORESCEINS; IMMUNITY, HUMORAL; IMMUNIZATION; MICE; SUCCINIMIDES; TH1 CELLS; TH17 CELLS;

EID: 85027951959     PISSN: None     EISSN: 15736830     Source Type: Journal    
DOI: 10.1007/s10571-011-9680-7     Document Type: Article

References (0)