Reduction in hypoxia-derived neuroinflammation and dysfunctional glutamate transporters by minocycline may restore hypoxia-injured cognition of neonatal rat

Sheng li xue bao : [Acta physiologica Sinica]

Volumn 68, Issue 2, 2016, Pages 148-156

  Li, Hong Chun ^a   Xiao, Jie ^a   Huang, Yi Long ^a   Li, Long Jun ^a   Jiang, Hong ^a   Huang, Li Xuan ^a   Yang, Ting ^a   Yang, Ling ^a   Li, Fan ^a  

^a KUNMING MEDICAL UNIVERSITY   (China)

Author keywords

[No Author keywords available]

Indexed keywords

GLUTAMATE TRANSPORTER; GLUTAMIC ACID DERIVATIVE; MINOCYCLINE; TAU PROTEIN; TRANSFORMING GROWTH FACTOR BETA1; TUMOR NECROSIS FACTOR;

ANIMAL; COGNITION; COGNITIVE DEFECT; DISEASE MODEL; HIPPOCAMPUS; HYPOXIA; INFLAMMATION; LEARNING; MEMORY; MEMORY DISORDER; NEWBORN; PHOSPHORYLATION; RAT;

AMINO ACID TRANSPORT SYSTEM X-AG; ANIMALS; ANIMALS, NEWBORN; COGNITION; COGNITION DISORDERS; DISEASE MODELS, ANIMAL; GLUTAMATES; HIPPOCAMPUS; HYPOXIA; INFLAMMATION; LEARNING; MEMORY; MEMORY DISORDERS; MINOCYCLINE; PHOSPHORYLATION; RATS; TAU PROTEINS; TRANSFORMING GROWTH FACTOR BETA1; TUMOR NECROSIS FACTOR-ALPHA;

EID: 85021389239     PISSN: 03710874     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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